myocytolysis


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my·o·cy·tol·y·sis

(mī'ō-sī-tol'i-sis),
Dissolution of muscle fiber.
[myo- + G. kytos, cell, + lysis, a loosening]

myocytolysis

/myo·cy·tol·y·sis/ (mi″o-si-tol´ĭ-sis) disintegration of muscle fibers.
coagulative myocytolysis  contraction band necrosis.

my·o·cy·tol·y·sis

(mī'ō-sī-tol'i-sis)
Dissolution of muscle fiber.
[myo- + G. kytos, cell, + lysis, a loosening]

myocytolysis

dissolution of muscle cells.
References in periodicals archive ?
Studies (64,66) have shown that uterine artery embolization causes hyaline (ischemic)-type necrosis, often associated with vacuolar change secondary to myocytolysis.
Cardiac sequelae including myocytolysis, serum enzyme elevations and arrhythmias are known to develop in a portion of stroke patients.
The main components of heart remodeling, which are independent of the characteristics of the cardiac pathological process, include [5-8] (1) changes in some of the cardiomyocytes (the disorder of the heart impulse-contraction process, [beta]-adrenergic desensitization, hypertrophy of cardiomyocytes, myocytolysis, impairment of the cytoskeleton proteins, etc.
2010) after injecting venom were myocytolysis (lysis of myocytes), coagulation necrosis, focal hemorrhage, thrombus formation both in myocardium and on endocardial surfaces.
3% of the patients, suggesting that the electrophysiology of cardial myocites can be altered before myocytolysis occurs (12).
Free radicals are toxic to the myocardium and can cause tissue damage that leads to extensive necrosis, myocytolysis and cellular edema86 Atherosclerotic plaque formation may be a reflection of sub-optimal GPx4 activity in the prevention of LD Loxidatio, with subsequent up take by endothelial cells and macrophages in arterial blood vessels91.
At autopsy, the heart showed left ventricular hypertrophy with widespread myocytolysis and an inflammatory cell infiltrate - findings in keeping with a catecholamine-induced cardiomyopathy.
The main feature of the syndrome is myocytolysis in both skeletal and cardiac muscles.
Sustained sympathetic stimulation can lead to heart damage, which begins as focal myocytolysis related to sustained levels of catecholamines (Horntagl et al.
All stages of ischemic injury were documented among the cases: coagulative necrosis and neutrophilic infiltrate in the acute phase, myocytolysis and granulation tissue in the subacute phase, and fibrosis in the chronic phase.