, polyphenoloxidase, catechol oxidase, and oxidoreductase), a copper-containing enzyme that induces formation of catecholamine quinone derivatives by its oxidase activity, makes a point since it has been demonstrated to cause a neuronal toxicity through oxidation of cytosolic excess dopamine in PD .
Laden and Porter (2001) have shown that resveratrol reduced cholesterol synthesis by inhibiting squalene monooxygenase
in vitro, a rate-limiting enzyme in cholesterol biosynthesis.
Silencing a cotton bollworm P450 monooxygenase
gene by plant-mediated RNAi impairs larval tolerance of gossypol.
Relative AOA and AOB abundance was estimated from ammonia monooxygenase
(amoA) gene copy numbers using real-time quantitative polymerase chain reaction (qPCR), as described by O'Sullivan et al.
or glycosyltransferase activities (Delye et al.
Pesticide tolerance of natural enemies may further be associated to ethion metabolization by cytochrome P450-dependent monooxygenase
A semi-quantitative cytochrome P450IA1 ELISA: a simple method for studying the monooxygenase
induction response in environmental monitoring and ecotoxicological testing of fish.
Mutation of the glucosinolate biosynthesis enzyme cytochrome P45083A1 monooxygenase
iIncreases camalexin accumulation and powdery mildew resistance.
ALKM-1 serve as a serologic marker for AIH type 2, and it reacts with recombinant antigen cytochromic monooxygenase
TMAO formation in both animal models and humans requires gut microbes (8-11), and proceeds via gut microbe metabolism of either choline, betaine, or other trimethylamine (TMA) nutrients to initially form TMA, which is delivered to the liver via the portal circulation where it is rapidly converted by host hepatic flavin monooxygenase
3 into TMAO (8, 12-14).
It is suggested that genetic polymorphisms in the enzymes of estrogen metabolism such as CYP1A1 and CYP1B1 (the main enzymes of monooxygenase
family of cytochrome P450) may affect the pathogenesis of leiomyoma .
Field-collected female adults consistently exhibited the highest monooxygenase
and Glutathione-S-transferases activities, responsible by mechanism is metabolic detoxification of pyrethroids.