mitochondrial biogenesis

mitochondrial biogenesis

the process by which mitochondria increase their ability to make adenosine triphosphate by synthesizing additional respiratory enzyme complexes.
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That is, exercise creates microRNA that decreases the number of immune cells that attack the muscle and heals the muscle by increasing aerobic metabolism through mitochondrial biogenesis.
Sodium hydrosulfide ameliorates oxidative stress and improves mitochondrial biogenesis in neonatal mouse cardiomyocytes
Transcription and replication of mitochondrial DNA (mtDNA) play a critical role in mitochondrial biogenesis during development (27).
Others discuss DNA self-catalyzed depurination, B12-dependent photoreceptors, engineering and in vivo applications of riboswitches, cyclic GMP-AMP as an endogenous second messenger, antibiotics, surface-active and surfactant proteins, functionally diverse lipid-binding compartments, the evolution of membrane coating complexes, endoplasmic reticulum-plasma membrane contact sites, mitochondrial biogenesis, oxidative stress, mammalian peroxiredoxins, redox-based regulation of bacterial behavior, heme uptake and bacterial cell membranes, biological probes built from fluoresceins and rhodamines, microbial rhodopsins, and cellular electron cryotomography.
SIRT1 gene also knowns as 'longevity gene', products a protein deacetylase that has been reported to suppress cardiovascular pathologies such as myocardial infarction via anti-apoptosis, anti-inflammation, or increasing mitochondrial biogenesis in model organisms.
Post infarction is followed by a state of increased insulin resistance, reduced fatty acid oxidation, and impaired mitochondrial biogenesis.
While there are multiple genes involved in athletic performance, given the association of PPARGC1A to mitochondrial biogenesis and conversion slow-twitch type I muscle fibers, this novel SNP may explain adaption in aerobic metabolism.
PQQ has been shown to promote mitochondrial biogenesis (creation of new mitochondria), which is essential for older cells to retain youthful energy output.
These free radicals result in molecular damage including lipid, protein, and DNA, leading to suppression of mitochondrial biogenesis and increased cellular damage (16).
The enzyme triumvirate, PGCla-SIRTl-AMPK plays a crucial role in mitochondrial biogenesis and resveratrol aids greatly in the process by enhancing the expression of these three enzymes.
I recently demonstrated that the promotion of mitochondrial biogenesis (MB) and fatty acid oxidation (FAO) in CD8+ T cells during an immune response is crucial for their development into long-lived TM cells.

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