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hyperchromasia |
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Injury to the respiratory cells resulted in distortion of the alveolar architecture such as morphologic changes characterized by cell flattening/stretching, hyperchromasia, and cellular death. Lesions with foci of cells displaying nuclear pleomorphism, hyperchromasia, prominent nucleoli, irregular nuclear borders, and/or altered nuclear to cytoplasmic ratios were considered dysplastic irrespective of their tinctorial characteristics. After 72 hours the PA treated cells showed evidence of scant cytoplasm and hyperchromasia when compared to control. |
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