gluconeogenesis


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Related to gluconeogenesis: Cori cycle

gluconeogenesis

 [gloo″ko-ne″o-jen´ĕ-sis]
the synthesis of glucose from noncarbohydrate sources, such as amino acids and glycerol. It occurs primarily in the liver and kidneys whenever the supply of carbohydrates is insufficient to meet the body's energy needs. Gluconeogenesis is stimulated by cortisol and other glucocorticoids and by the thyroid hormone thyroxine. Formerly called glyconeogenesis.

glu·co·ne·o·gen·e·sis

(glū'kō-nē'ō-jen'ĕ-sis),
The formation of glucose from noncarbohydrates, such as protein or fat.
Synonym(s): glyconeogenesis (2)

gluconeogenesis

/glu·co·neo·gen·e·sis/ (gloo″ko-ne″o-jen´ĕ-sis) the synthesis of glucose from molecules that are not carbohydrates, such as amino and fatty acids.

gluconeogenesis

(glo͞o′kə-nē′ə-jĕn′ĭ-sĭs)
n.
The formation of glucose, especially by the liver, from noncarbohydrate sources, such as amino acids and the glycerol portion of fats.

glu′co·ne′o·ge·net′ic (-ō-jə-nĕt′ĭk) adj.

gluconeogenesis

[glo̅o̅′kōnē′ōjen′əsis]
the formation of glucose from glycerol and proteins rather than from carbohydrates. Also called glyconeogenesis.
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Gluconeogenesis

gluconeogenesis

The formation of glucose from noncarbohydrate molecules–eg, amino acids, lactic acid

glu·co·ne·o·gen·e·sis

(glū'kō-nē'ō-jen'ĕ-sis)
The formation of glucose from noncarbohydrates, such as protein or fat.
Compare: glyconeogenesis

gluconeogenesis

The formation of glucose from non-carbohydrate sources, especially from AMINO ACIDS from protein. GLUCOCORTICOID hormones stimulate gluconeogenesis.

gluconeogenesis

the process by which PYRUVIC ACID (pyruvate) is converted to GLUCOSE. This is not the exact reversal of GLYCOLYSIS. Three of the reactions of glycolysis are irreversible and it is in these three that gluconeogenesis differs. In the CELL gluconeogenesis is normally more active when there is little need for ATP. The process meets the needs of the body for glucose when CARBOHYDRATE is not available in adequate amounts from the diet. Non-carbohydrates, such as FAT and PROTEIN, can be converted into glucose, notably in the LIVER and KIDNEY.

gluconeogenesis

synthesis of glucose from non-carbohydrate precursors mainly in the liver and to a smaller extent in the renal cortex. Precursors include pyruvate, lactate, glycerol and the glucogenic amino acids derived from skeletal muscle. The glucose-alanine cycle involves the conversion of alanine, formed in muscle, to glucose in the liver; activity of the cycle is increased during the postabsorptive state and in starvation or prolonged exercise, slowly mobilizing glycogen stores and using protein for the maintenance of normal blood glucose concentration. Gluconeogenesis and export of glucose from the liver are promoted by the hormone glucagon, and inhibited by insulin.

glyconeogenesis

formation of glycogen from non-carbohydrate sources; occurs in non-controlled diabetes mellitus (body protein and fat breakdown yield acetone and ketone bodies, used as an alternative cellular energy source)

glu·co·ne·o·gen·e·sis

(glū'kō-nē'ō-jen'ĕ-sis)
The formation of glucose from noncarbohydrates, such as protein or fat.

gluconeogenesis

(gloo´kōnē´ōjen´əsis),
n the formation of glycogen or glucose from noncarbohydrate sources (e.g., the glycogenic amino acids, glycerol, lactate, and pyruvate) by pathways mainly involving the citric acid cycle and glycolysis.

gluconeogenesis

the synthesis of glucose from noncarbohydrate sources, such as amino acids, propionate and glycerol. It occurs primarily in the liver and kidneys whenever the supply of carbohydrates is insufficient to meet the body's metabolic demands or in the rumen by the action of bacteria in well-fed ruminants. Gluconeogenesis is stimulated by cortisol and other glucocorticoids and by glucagon. Formerly called glyconeogenesis.
References in periodicals archive ?
Marein improved the insulin resistance induced by high glucose concentrations in HepG2 cells via CaMKK/AMPK/GLUTl to promote glucose uptake, via IRS/Akt/GSK-3[beta] to increase glycogen synthesis, and via Akt/FoxO1 to decrease gluconeogenesis.
ATP amount (x) resulting from glyoxysomal degradation of fatty acids with carbon number multiple of 4, followed by gluconeogenesis until phosphoenolpyruvate and its catabolism, to produce energy, may be calculated by the sum of ATP rates produced and consumed in [beta]-oxidation ([ATP.
Moreover, we found that the regulation of liver gluconeogenesis and glycogenolysis and islet insulin secretion were markedly impaired by arsenic exposure in estrogen-deficient female mice, which could be effectively reversed by estrogen supplementation.
Gluconeogenesis in hepatopancreas of Chasmagnathus granulala crabs maintained on high-protein or carbohydrate-rich diets.
The release of pyridoxal phosphate during exercise is probably related to gluconeogenesis in the liver (1).
Increasing FFA concentration reduce the inhibiting effect of insulin upon the gluconeogenesis and glycogenolysis, hereby the blood glucose level increased.
Review and an experimental study on the physiologicaland quantitative aspects of gluconeogenesis in lactating ruminants.
As glucose metabolism in ruminants differs from that of nonruminants because circulating glucose is mainly provided by gluconeogenesis in the liver from precursors such as propionic acid or glucogenic amino acids.
The reaction is reversed in gluconeogenesis using fructose-1,6-bisphosphatase: fructose-1,6-bisphosphate + [H.
Dr Triggle explained that it has generally been assumed that the beneficial effects of metformin are linked to its ability to inhibit hepatic gluconeogenesis (generation of glucose from non-carbohydrate sources by the liver), which means less glucose in the blood and therefore less vascular deterioration.
Dr Triggle said that it has generally been assumed that the beneficial effects of metformin are linked to its ability to inhibit hepatic gluconeogenesis (generation of glucose from non-carbohydrate sources by the liver), which means less glucose in the blood and therefore less vascular deterioration.