glomerulonephritides

glomerulonephritides

Plural of glomerulonephritis.
References in periodicals archive ?
4-7) In the latter scenario, similar lesions can be seen in other glomerulonephritides, such as, light-chain deposition disease, amyloidosis, immunotactoid nephropathy, the membranoproliferative form of diffuse glomerulonephritis, and idiopathic, nodular glomerulosclerosis.
They first relate the top 100 secrets in the field, then describe patient assessment, acute kidney injury, chronic kidney disease, primary and secondary glomerular disorders, infection-associated glomerulonephritides, other renal parenchymal diseases, diseases in special populations like pregnant women and the elderly, treatment options, transplantation, hypertension, and acid-base and electrolyte disorders.
Sunday speakers: Robert Rountree; Alex Vasquez, DC, ND, DO; and Patrick Donovan, RN, ND, Topics Include naturopathic approach to autoimmune glomerulonephritides, autoimmune thyroid disease, autoimmune hepatitis, idiopathic thrombocytic purpura, and other autoimmune conditions; and vascular, renal, & cardiac complications of autoimmunity and systemic inflammation.
IgA nephropathy, the most common cause of renal failure among the primary glomerulonephritides, is characterized by deposition of IgA antibodies in the glomerulus.
3% (all with HIVAN) Other non-glomerulonephritic renal diseases - Other glomerulonephritides - Post-infectious glomerulonephritis - Mesa ngioproliferative glomerulonephritis - IgA nephropathy - Membranoproliferative glomerulonephritis 6.
9] More severe or progressive renal injury, such as that caused by the glomerulonephritides, focal segmental glomerulosclerosis, and hemolytic uremic syndrome, causes loss of the glomerular ability to discriminate between proteins by si ze, which results in nonselective proteniuria.
28) The magnitude of the nephritogenic potential of macrophage infiltration in proliferative glomerulonephritides, being highest in lupus nephritis and cryoglobulinemic GN, was demonstrated by Ferrario et al.
Furthermore, these data show that PDGF-D is over-expressed in mesangioproliferative states and can act as an auto-, para- or even endocrine glomerular cell mitogen, indicating that antagonism of PDGF-D may represent a novel therapeutic approach for the treatment of mesangioproliferative glomerulonephritides.
There is a consensus among renal pathologists that immune complex deposition is a diffuse phenomenon, meaning that if immune complex glomerulonephritis occurs, all glomeruli with open capillaries will show immune complex deposits, even if the light microscopic lesions are focal (in many glomerulonephritides not all glomeruli show light microscopic abnormalities).