gingival fibromatosis

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Related to gingival fibromatosis: Gingival hyperplasia


1. the presence of multiple fibromas.
2. the formation of a fibrous tumorlike nodule arising from the deep fascia, with a tendency to local recurrence.
fibromatosis gingi´vae (gingival fibromatosis) a noninflammatory fibrous hyperplasia of the gingivae and palate, manifested as a dense, smooth, or nodular overgrowth of the tissues. It is usually inherited as an autosomal dominant trait, but some cases are idiopathic and others are produced by drugs.
palmar fibromatosis fibromatosis involving the palmar fascia, and resulting in Dupuytren's contracture.
plantar fibromatosis fibromatosis involving the plantar fascia manifested as single or multiple nodular swellings, sometimes accompanied by pain but usually unassociated with contractures.

gin·gi·val fi·bro·ma·to·sis

fibromatosis that may be associated with trichodiscomas. Several genetic forms are known, all autosomal dominant [MIM*135300, *135400, *135500, *135550].

gin·gi·val fib·ro·ma·to·sis

(jinji-văl fībrō-mă-tōsis)
Disorder that may be associated with trichodiscomas.

gin·gi·val fib·ro·ma·to·sis

(jinji-văl fībrō-mă-tōsis)
Fibromatosis that may be associated with trichodiscomas. Several genetic forms are known.
References in periodicals archive ?
Hereditary gingival fibromatosis as an isolated trait is usually transmitted
Other genetic loci such as 8, 14q, 19p, 19q and Xq are also related to syndromes associated with hereditary gingival fibromatosis.
As mentioned earlier, hereditary gingival fibromatosis depicts the generalized gingival enlargement with no other systemic manifestations.
Oro-facial granulomatosis can be demarcated from hereditary gingival fibromatosis by observing the features of oro-facial granulomatosis in addition to gingival enlargement which are absent in hereditary gingival fibromatosis.
Such features are absent in Hereditary gingival fibromatosis.
Keeping all these facts in mind, diagnosis of hereditary gingival fibromatosis was made on the basis of positive family history, parental consanguinity, classical clinical features and absence of any syndromic association.