gastrins


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gas·trins

(gas'trinz),
Hormones secreted in the pyloric-antral mucosa of the mammalian stomach that stimulate secretion of HCl by the parietal cells of the gastric glands; there are three main types: big gastrin (34 aminoacyl residues), little gastrin (17 residues), and minigastrin (14 residues), as well as sulfated derivatives. The C-terminal pentapeptide is also seen in cholecystokinin and cerulein.
[G. gastēr, stomach, + -in]

gas·trins

(gas'trinz)
Hormones secreted in the pyloric-antral mucosa of the mammalian stomach that stimulate secretion of HCl by the parietal cells of the gastric glands; a competitive inhibitor of gastrins is cholecystokinin.
[G. gastēr, stomach, + -in]

gas·trins

(gas'trinz)
Hormones secreted in the pyloric-antral mucosa of the mammalian stomach.
[G. gastēr, stomach, + -in]
References in periodicals archive ?
Variations in the sulfation of circulating gastrins in gastrointestinal diseases.
Gastrin was the first gastrointestinal hormone to be measured in plasma (1-4).
Today, the picture is changed: Peptic ulcer is caused by Helicobacter pylori infection and does not need gastrin measurement for diagnosis.
In gastrinoma cells, progastrin is processed less efficiently than in normal gastrin cells (8,25).
Patterns of gastrin components in serum during feeding in normal subjects and duodenal ulcer patients.
Gel fltration studies on immunoreactive gastrin in serum from Zollinger-Ellison patients.
The plasma samples were stored at -20 [degrees]C until the measurement of gastrin and CCK.
Although several high-titer and high-affinity antisera were C-terminal directed (1559, 1560, 1564, 8007, and 8011), they all cross-reacted with gastrin peptides to a degree making them useless for plasma measurement.
Disturbed islet-cell function related to endogenous gastrin release.
Gastrin is a linear peptide hormone produced by gastrin cells, having regulatory functions of acute gastric acid secretion as well as the gastric epithelial cell population (Kovac et al.
ATPase, the secretion of acid and gastrin level, but had little effect on the mucosal repair and the activity of pepsin in a PL induced ulcer.
ATPase activity and the secretion of gastrin and gastric acid in PL-induced rats.