fatty streak


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Related to fatty streak: Foam cells

fatty streak

Early evidence of atherosclerosis, in which cholesterol, macrophages, and smooth muscle cells accumulate in the intima of arteries.
See: atherosclerosis
See also: streak

fatty streak

fibrosed extracellular lipid at the intimal surface of an artery causing local stenosis and roughening; earliest manifestation of arterial or arteriolar pathology, culminating as atheroma formation, typically presenting at arterial bifurcations
References in periodicals archive ?
There is a widespread consensus that the more fatty streaks you have, and the thicker they get, the more likely they are to go on to fibrous plaques," says McGill.
A macroscopically 'normal' aorta was defined as one in which the intimal surface of the vessel was smooth, glossy and of uniform colour, with no fatty streaks or other lesions.
He explained the procedure: "The hamsters were on a high-fat diet and you get signs of heart disease with the fatty streaks in the arteries after 12 weeks.
A glass a day can protect your heart by stopping the production of endothelin-1, responsible for the fatty streaks in arteries.
Furthermore, several studies have shown that fetuses and children born of hypercholesterolemic mothers have an increased incidence of fatty streaks in the aorta.
The fruit also hampers hardening of the arteries by fatty streaks along blood vessel walls (arteriosclerosis) by helping to stop high cholesterol.
pneumoniae antigens or DNA in intimal thickening and fatty streaks of young adults and Alaskan Natives (the latter group at low risk for coronary atherosclerosis) supports an early microbial role in pathogenesis (30).
If you eat lots of greasy foods, ugly fatty streaks may already be building up in your arteries.
Claudio Napoli and colleagues showed in a previous study that fatty streaks, the earliest lesions of atherosclerosis, have already formed in fetal arteries.
Inflammation, which is implicated in the formation of early fatty streaks as well as at the onset of adverse cardiovascular events, causes cells within the plaque to secrete enzymes that degrade and weaken the fibrous cap, leading to rupture of unstable plaques and thrombus (clot) formation.