familial combined hyperlipidemia


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familial combined hyperlipidemia

Metabolic disease A common–1:300 AD disorder with ↑ TGs and/or cholesterol Lab ↑ apoB, ↑ LDL-C, ↑ VLDL-C, mild ↓ HDL-C, apoA1 Clinical CAD, first MI as early as age 40, overweight, HTN Management Diet–weight loss, exercise, lipid-lowering drugs; smoking is forbidden as it exacerbates ASHD
References in periodicals archive ?
Complex genetic contribution of the Apo AI-CIII-AIV gene cluster to familial combined hyperlipidemia.
Bimodality of plasma apolipoprotein B levels in familial combined hyperlipidemia.
Athyros VG, Papageorgiou AA, Hatzikonstandinou HA, et al: Safety and efficacy of long-term statin-fibrate combinations in patients with refractory familial combined hyperlipidemia.
Their topics include how much evidence is enough, epidemiological aspects of lipid and lipoprotein levels in relation to cardiovascular diseases, whether raising HDL protects against atherosclerosis, the optimal treatment of patients with familial combined hyperlipidemia, managing lipid and lipoprotein disorders in children, the link between chronic inflammatory diseases and cardiovascular risk, dealing with inborn errors in cholesterol and bile acid biosynthesis, and when to fear statin interactions.

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