palsy /pal·sy/ (pawl´ze) paralysis.
facial palsy
palsy (pôl´zē),
facial palsy
Bell's palsy unilateral facial paralysis of sudden onset due to a lesion of the facial nerve, resulting in characteristic facial distortion.
cerebral palsy any of a group of persisting qualitative motor disorders appearing in young children, resulting from brain damage caused by birth trauma or intrauterine pathology.
Erb's palsy , Erb-Duchenne palsy Erb-Duchenne paralysis.
facial palsy Bell's p.
progressive bulbar palsy chronic, progressive, generally fatal paralysis and atrophy of the muscles of the lips, tongue, mouth, pharynx, and larynx due to lesions of the motor nuclei of the lower brain stem, usually occurring in late adult years.
wasting palsy spinal muscular atrophy.
| facial palsy n. Unilateral paralysis of the facial muscles supplied by the facial nerve. Also called Bell's palsy, facial paralysis, facioplegia, prosopoplegia. |
facial palsy
Etymology: L, facies, face; Gk, paralyein, to be palsied
a loss of motor nerve function in the muscles of the face. See also Bell's palsy.
palsy (pôl´zē),
n a general term for
paralysis but preferred by some to refer to certain types of paralysis.
paralysis but preferred by some to refer to certain types of paralysis.
palsy, Bell's,
n.pr facial paralysis believed to result from inflammation in or around the facial nerve. One side of the face sags, the corner of the oral cavity droops, the eyelid does not close, and saliva dribbles from the corner of the oral cavity on the affected side. See also paralysis, facial.
palsy, cerebral,
n 1. a collective term for neurologic defects with associated disturbances of motor function. The disturbances vary in cause and anatomic type (e.g., acquired, hereditary, natal, postnatal, congenital palsy).
n 2. a nonspecific term representing a group of pathologic conditions having the following common, related characteristics: agenesis, or a lesion of nervous tissue within the cranium; interference with voluntary muscular movements; disabling disorders of a chronic nature, neither acute nor progressive; and occurrence of the original lesion at the date of birth of the patient or before the development of learned muscular function.
3. a condition caused by damage to the motor centers of the brain, resulting in varying disturbances of motor function and often accompanied by mental subnormality.
n 2. a nonspecific term representing a group of pathologic conditions having the following common, related characteristics: agenesis, or a lesion of nervous tissue within the cranium; interference with voluntary muscular movements; disabling disorders of a chronic nature, neither acute nor progressive; and occurrence of the original lesion at the date of birth of the patient or before the development of learned muscular function.
3. a condition caused by damage to the motor centers of the brain, resulting in varying disturbances of motor function and often accompanied by mental subnormality.
)
Bell's palsy.
palsy, creeping,
n See gait, spastic.
palsy, facial,
n paralysis of the muscles supplied by the seventh cranial nerve. It may be associated with peripheral lesions, neoplasms invading the temporal bone, acoustic neuromas, pontine disease, and herpes zoster involving the geniculate ganglion. Bilateral paralysis may occur in uveoparotid fever and polyneuritis.
palsy, lead,
n a weakness and paralysis of the hand, wrist, and fingers, associated with lead poisoning. See also lead (Pb).
facial palsy
Bell's palsy, cranial mononeuropathy VII, facial mononeuropathy, facial nerve palsy, facial neuralgia Neurology Acute peripheral paralysis of the face due to a herpes simplex immune-mediated condition often characterized by
severe pain in the trigeminal nerve Epidemiology Risk of FP ↑ with age; age 10 to 19, 2:1, ♀:♂; age 40, 3:2, ♂:♀; pregnant ♀ have 3.3 times ↑ risk than nonpregnant; DM = 4.5 times↑ risk of FP; 10% of Pts
have positive family Hx of FP Pathogenesis FP is due to reactivation of the virus leading to replication of virus within the ganglion cells; the virus travels down the axons, inducing inflammation Clinical Abrupt onset, drooping mouth, unblinking
eye, twisted nose, uneven smile, distorted expressions; paralysis hits maximum in 1 to 14 days; retroauricular pain, facial numbness, epiphora, parageusia, ↓ tearing, hyperacusis, hypoesthesia or dysesthesia of cranial nerves–CN V and IX,
motor paresis of CN IX and X, papillitis of tongue DiffDx, unilateral Tumors or masses, otitis media, sarcoid, Lyme disease, skull fracture, facial injury DiffDx, bilateral Guillain-Barré syndrome, Melkersson-Rosenthal syndrome, Möbius
syndrome, motor neuron disease, myasthenia gravis Etiology Trauma, Bell's palsy, stroke, parotid tumors, intracranial tumors Management Microvascular and micro-neurosurgical tissue transfers allow restoration of functional, unconscious,
symmetrical facial movements, acyclovir, steroids–efficacy is uncertain, artificial tears, neuromuscular retraining–eg, mirror/visual feedback, biofeedback or electromyography feedback Prognosis 60 to 80% recover, especially if
incomplete paralysis, and Pt is young
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