exertional rhabdomyolysis


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ex·er·tion·al rhab·do·my·ol·y·sis

rhabdomyolysis produced in those susceptible by muscular exercise.
Episodic haemoglobinuria due to haemolysis caused by repeated mechanical injury to red cells that travel through small vessels overlying the bones of hands and feet in long-distance marching—soldiers, marathon running, calisthenics, karate
Lab Myoglobinuria, proteinuria, increased BUN, increased enzymes—e.g., creatinine phosphokinase, increased lactic acid

exertional

emanating from or pertaining to exertion.

exertional rhabdomyolysis
occurs as an acute recumbency or immobility state when muscle masses disrupt during exercise. Occurs as equine paralytic myoglobinuria in horses after unaccustomed exercise while on a heavy carbohydrate diet, in sheep and cattle after violent physical exercise, in racing greyhounds and as capture or transport myopathy in deer and other wildlife. Called also azoturia, Monday morning disease, tying up syndrome, capture myopathy.

rhabdomyolysis

disintegration of striated muscle fibers with excretion of myoglobin in the urine.

equine rhabdomyolysis
see paralytic myoglobinuria.
exertional rhabdomyolysis
see exertional rhabdomyolysis.
transient exertional rhabdomyolysis
References in periodicals archive ?
However, there is also concern that athletes with SCT have an elevated risk of exercise-related sudden death secondary to exertional heat illness or acute exertional rhabdomyolysis (Eichner 2010).
Exertional rhabdomyolysis cases mostly involve individuals who were inexperienced exercisers, dehydrated and uneducated in fitness (Sandhu et al.
Eccentric muscle damage response can be more stressed in individuals that are susceptible toward exertional rhabdomyolysis (high responders).
In summary, exertional rhabdomyolysis localized to one muscle is not a rare condition.
2001) Exertional rhabdomyolysis in a body builder abusing anabolic androgenic steroids.
Exertional rhabdomyolysis is accompanied by unusually intense muscle pain.
Exertional rhabdomyolysis may be linked to the pathogenesis of EAH with release of muscle-derived interleukin(IL)-6 leading in part to a decrease in urine formation.
former Marine Corps Marathon medical director; and exertional rhabdomyolysis by Bruce Adams, M.
In contrast to nonspecific elevations of myoglobin and creatine kinase MB (CK-MB) in athletes due to exertional rhabdomyolysis of trained skeletal muscle(1,2), reported increases in cardiospecific troponins after competition may indicate silent injury to the myocardium (3,4,5,6,7,8).
Contents in Brief: In contrast to non-specific post-race elevations in myoglobin and CK-MB as early-stage markers for AMI due to exertional rhabdomyolysis of trained skeletal muscle, late-stage markers including cTnI and cTnT remained negative by rapid quantitative methods within 24 hours after competition.