erythroid


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Related to erythroid: erythroid cells, Erythroid progenitor cells

erythroid

 [er´ĭ-throid]
1. of a red color; reddish.
2. pertaining to any of the cells in the erythrocytic series.

er·y·throid

(er'i-throyd, ĕ-rith'royd),
Reddish in color.

erythroid

/er·y·throid/ (er´ĭ-throid)
1. of a red color; reddish.
2. pertaining to the cells of the erythrocytic series.

erythroid

[erith′roid]
1 reddish in color.
2 pertaining to erythrocytes.

er·y·throid

(e-rith'royd)
Of a reddish color.

erythroid

1. of a red color; reddish.
2. pertaining to the developmental series of cells ending in erythrocytes.

erythroid aplasia
see pure red cell aplasia.
burst forming unit-erythroid
produced in in vitro cultures of erythroid stem cells. It has low sensitivity to erythropoietin and gives rise to colony forming unit-erythroid which, with erythropoietin, gives rise to erythroid cells.
erythroid hypoplasia
see hypoplastic anemia.
erythroid leukosis
see avian erythroblastosis.
References in periodicals archive ?
In one of the largest studies to date, Voulgarelis et al (10) looked at 40 cases of SLE and similarly found frequent dysplastic changes, most notable in the erythroid and megakaryocytic lineages, as well as ALIP that showed striking similarity to the control cases of MDS, refractory anemia subtype.
An erythroid chaperone that facilitates folding of [alpha]-globin subunits for hemoglobin synthesis.
In infection, cytokines and their products therefore cause: (i) a diversion of iron into iron stores in the RES, resulting in decreased iron concentration in the plasma, thus limiting its availability to red cells for haemoglobin synthesis; (ii) an inhibition of erythroid progenitor cell proliferation; and (iii) inappropriate production and activity of Epo.
Firstly, it is directly toxic to developing cells, causing an increased or high-normal MCV and vacuolation of erythroid precursors.
The ineffective hematopoiesis results in megaloblastoid changes of the erythroid precursors in the bone marrow.
Bianchi N, Chiarabelli C, Borgatti M, Mischiati C, Fibach E, Gambari R (2001) Accumulation of gamma-globin mRNA and induction of erythroid differentiation after treatment of human leukaemic K562 cells with tallimustine.
In a presentation titled, "From GWAS to the Clinic: Genome-Editing the Human Bcl11a Erythroid Enhancer for Fetal Globin Elevation in the Hemoglobinopathies" (Abstract #53), new data were described demonstrating the effectiveness of knocking out the BCL11A Enhancer in hematopoietic stem and progenitor cells (HPSCs) as an approach to elevate fetal globin in a manner that is specific for red blood cell precursors (erythroid-specific), thereby eliminating any effect of reduced BCL11A expression in HSPCs or other non-erythroid lineages.
The erythroid lineage demonstrated left-shifted maturation with increased numbers of pronormoblasts and basophilic normoblasts, many of which contained multiple small cytoplasmic vacuoles.
Analysis of bone marrow aspirate showed cellular marrow with normal granulopoiesis and megakaryocytopoiesis, but the almost complete absence of erythroid precursor cells.
Hematopoietic tissue included megakaryocytes, maturing granulocytes, lymphocytes and erythroid precursors (Fig.
NF-E2 is expressed only in erythroid cells, megakaryocytes, and mast cells.
The major issue is that the bone marrow erythroid mass fails to expand appropriately in response to anaemia.

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