epistatic


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Related to epistatic: dominant epistasis, modifier genes

ep·i·stat·ic

(ep'is-tat'ik),
Relating to epistasis.

ep·i·stat·ic

(ep'i-stat'ik)
Relating to epistasis.
References in periodicals archive ?
In present studies, in cross Hashim-08 X LU-26, the AUDPC was controlled by mixed two additive dominant epistatic major genes plus additive-dominant epistasis of polygene which revealed that AUDPC was mainly governed by dominant effects of the major genes with epistatic nature with polygene.
Our study also revealed that VDR rs1544410 and VDR rs2228570 are in epistatic interaction, and the VDR rs1544410_rs2228570 haplotype AC is associated with dyslipidemia in HD patients.
It is being proposed that a risk score comprising the most significantly associated variants for each blood pressure phenotype may be developed and evaluated for epistatic effect amounts to several millimeters mercury of blood pressure (13).
Full-sib experiments cannot partition additive genetic variance from dominance or epistatic genetic variance.
The authors suggested that the changes in intermediate ratios through the pathway might be the foundation for observed statistical epistatic interactions.
2]-derived NILs were used to verify the main effects of QTL-M, QTL-H, and QTL-G, and to evaluate the epistatic interactions among the QTLs in a Benning genetic background (Table 1).
The results indicate that the effect of inbreeding on the interaction between host and pathogen in this pathosystem is unpredictable at the local population level, because: (1) strong genotypic differences in inbreeding effekt exist for both active(and passive resistance, making the effect of inbreeding at the population level dependent on the genot ypic composition of the (founder) population; (2) effects of inbreeding on active and passive resistance were not correlated, making the net effect of inbreeding on field resistance unpredictable; and (3) in several lines, evidence for epistatic effects was found, making the effect of inbreeding dependent on the actual inbreeding level of the genotype.
However, major disputes have arisen about which kind of plasticity gene is more important in determining the norms of reaction, and whether the epistatic gene action of regulatory genes can cause the evolution of plastic responses (Scheiner 1993; Schlichting and Pigliucci 1993; Via 1993).
Hallauer (1990) reported that since inbreeding is conducted simultaneously with hybrid evaluations, favorable epistatic gene combinations can ultimately be fixed in the inbred lines.
Moreover, in a large-scale perturbation study, we will specifically address the role of the kinome in antiviral innate immune signalling pathways, exploring the role of its individual members and their epistatic relationships in orchestrating gene expression.