epiphyseal

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ep·i·phys·i·al

, epiphyseal (ep'i-fiz'ē-ăl), Avoid the mispronunciation epiphyse'al.
Relating to an epiphysis.

epiphyseal

/epi·phys·e·al/ (ep″ĭ-fiz´e-al) pertaining to or of the nature of an epiphysis.

epiphyseal

[ep′ifiz′ē·əl, ipif′əsē′əl]
Etymology: Gk, epi, above, phyein, to grow
pertaining to or resembling the epiphysis. Also spelled epiphysial.

epiphyseal

emanating from or pertaining to the epiphysis.

epiphyseal aseptic necrosis
caused by (1) idiopathic primary necrosis of the epiphysis in growing small-breed dogs (Legg-Calvé-Perthes disease); (2) fracture of the femoral neck; or (3) epiphyseal slippage, particularly of the femoral head in young dogs, cats, pigs, calves and foals. The disease has a characteristic radiographic appearance.
epiphyseal cartilage
between the epiphysis and the diaphysis of long bones; growth at the cartilage is responsible for continuing growth of the bone; when growth ceases the cartilage disappears. Called also growth plate, physis.
epiphyseal detachment
epiphyseal dysplasia
an inherited defect of dogs characterized by very short limbs and early degenerative arthropathy. Called also chondrodystrophia fetalis and pseudoachondroplastic dysplasia of Miniature poodles. A similar histological lesion occurs in multiple epiphyseal dysplasia in Beagles.
epiphyseal fracture
one involving the epiphysis. See also salter classification.
epiphyseal plate
the thin plate of cartilage between the epiphysis and the shaft of a long bone; it is the site of growth in length and is obliterated by epiphyseal closure. Called also growth plate, physis.
epiphyseal scar
on radiographs, the radiodense band seen at the junction of the epiphysis and metaphysis, which represents the closed physis.
References in periodicals archive ?
We observed significant abnormalities in epiphyseal cartilage matrix and, as a consequence, in endochondral bone formation.
Consistent with the turning of the epiphyseal cartilage into a mosaic of discrete chondrons separated by clefts and fibroblast-like cells, immunohistology indicates the absence of a chemically recognizable decorin-enriched interterritorial matrix; in contrast, the biglycan-enriched territorial matrix was preserved.
Although mutations in the diastrophic dysplasia sulfate transporter gene are expected to result in complex and multiple chemical abnormalities of the cartilage matrix, the immunohistochemical distribution of biglycan and decorin discloses a major structural change in the overall organization of epiphyseal cartilage matrix, with important implications for the pathogenesis of the abnormal growth and modeling of endochondrally formed bones in ACG-IB.
05 Table 3: Morphometric characteristics of epiphyseal cartilage in long tubular bones of albino rats at different terms of postnatal development in normal animals and those whose mothers were exposed to the lead acetate.