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hyperoxaluria
(redirected from enteric hyperoxaluria)

   Also found in: Wikipedia 0.02 sec.
hyperoxaluria /hy·per·ox·al·uria/ (-ok″sah-lu″re-ah) an excess of oxalates in the urine.
enteric hyperoxaluria  formation of calcium oxalate calculi in the urinary tract after resection or disease of the ileum, due to excessive absorption of oxalate from the colon.
primary hyperoxaluria  an inborn error of metabolism with defective glyoxylate metabolism, excessive urinary excretion of oxalate, nephrolithiasis, nephrocalcinosis, early onset of renal failure, and often a generalized deposit of calcium oxalate.

hy·per·ox·al·u·ri·a (hpr-ks-lr-)
n.
The presence of an unusually large amount of oxalic acid or oxalates in the urine.

hyperoxaluria
[-ok′səloo͡r′ē·ə]
an excessive level of oxalic acid or oxalates, primarily calcium oxalate, in the urine. The cause is usually an inherited deficiency of an enzyme needed to metabolize oxalic acid, which is present in many fruits and vegetables, or a disorder of fat absorption in the small intestine. An excess of oxalates may lead to the formation of renal calculi and renal failure. Treatments include pyridoxine, forced fluids, and a low-oxalate diet.

hyperoxaluria (hī´prok´slr´ē),
n an excessive level of oxalic acid or oxalates, primarily calcium oxalate, in the urine. The cause is usually an inherited deficiency of an enzyme needed to metabolize oxalic acid, which is present in many fruits and vegetables, or a disorder of fat absorption in the small intestine. An excess of oxalates may lead to the formation of renal calculi. Treatment may include pyridoxine, forced fluid, and a low-oxalate diet.
hyperoxaluria, primary,
n an inherited deficiency of the enzyme that metabolizes oxalic acid, resulting in an excessive level of oxalic acid or oxalates in the urine.

hyperoxaluria
an excess of oxalate in the urine; occurs in dogs in association with oxalate urolith formation.

primary hyperoxaluria
inherited metabolic defect in cats. Characterized by heavy deposits of oxalates in renal tubules, leading to oxalate nephrosis and fatal uremia before the patient reaches a year of age.


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We concluded that the patient had enteric hyperoxaluria due to excess oxalate absorption in his colon leading to subsequent excretion in his urine.
ALTU-237 is an oral form of an oxalate-degrading enzyme for the treatment of primary hyperoxaluria and enteric hyperoxaluria, as well as to prevent the recurrence of kidney stones.
There are no current effective pharmacological treatments for primary hyperoxaluria, enteric hyperoxaluria or recurrent kidney stones.
 
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