endotoxemia


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endotoxemia

 [en″do-tok-se´me-ah]
the presence of endotoxins in the blood.

en·do·tox·e·mi·a

(en'dō-tok-sē'mē-ă),
Presence in the blood of endotoxins, which, if derived from gram-negative rod-shaped bacteria, may cause a generalized Shwartzman phenomenon with shock.

endotoxemia

/en·do·tox·e·mia/ (en″do-toks-ēm´e-ah) the presence of endotoxins in the blood, which may result in shock.

en·do·tox·e·mi·a

(en'dō-tok-sē'mē-ă)
Presence in the blood of endotoxins, which, if derived from gram-negative, rod-shaped bacteria, may cause a generalized Shwartzman phenomenon with shock.
Synonym(s): endotoxaemia.

endotoxemia

the presence of endotoxins in the blood.
References in periodicals archive ?
It is well known that endotoxemia leads to excessive production of the vasodilator NO via iNOS (31,32).
Los niveles plasmaticos de LPS son particularmente importantes en algunas enfermedades como la sepsis, en las cuales se ha observado que el receptor para las HDL, SR-B1, controla la respuesta frente a la endotoxemia, reduciendo la activacion de NF-[kappa]B (44).
Enterobacteriaceae members are known to produce alcohol as a fermentation product, which promotes gut permeability and leads to metabolic endotoxemia (18).
There are several etiologic factors involved in the onset of gastrointestinal symptoms during exercise, such as intestinal hypoperfusion endotoxemia, dehydration, diet, mechanical factors, as well as aspects related to the exercise, which possibly explains the symptomatic prevalence observed in this study.
The effects of isoflurane anesthesia and mechanical ventilation on renal function during endotoxemia.
It has been reported that tissue damage and endotoxemia related to obstructive jaundice causes an increase in the production of oxygen free-radicals which in turn increases lipid peroxidation (3, 4).
Evidence that chronic alcohol exposure promotes intestinal oxidative stress, intestinal hyperpermeability and endotoxemia prior to development of alcoholic steatohepatitis in rats.
However, evidence presented in the last two decades suggests that heat- and exercise-induced endotoxemia and the downstream consequences of systemic inflammation may play important roles in triggering and driving the mechanisms of heat stroke [2,7-9].
Las propiedades pro aterogenicas de las lipoproteinas de muy baja densidad VLDL en relacion al lipopolisacarido bacteriano podrian tener un efecto potenciado como respuesta a la endotoxemia causada por la terapia inicial, se necesitaria un periodo de seguimiento mayor para evaluar cambios en estos niveles.
Endotoxemia and function failure of intestinal mucosal barrier may lead to exacerbation of patients with hepatic cirrhosis.
Case reports of endotoxemia associated with the use of contaminated propofol have also been published (5,34,35).