diabetic microangiopathy

diabetic microangiopathy

Microvascular disease Any clinical or pathological changes resulting from small vessel disease in PTs with DM Measurement Capillary HTN can be measured directly by microcannulation of nailfold capillaries with a glass micropipette Prognosis Progression can be slowed by tight control of serum glucose levels, especially with long-term intense insulin therapy
References in periodicals archive ?
Diabetic microangiopathy is an independent predictor of incident diabetic foot ulcer.
Potential role of curcumin phytosome (Meriva) in controlling the evolution of diabetic microangiopathy.
sup][5] Diabetic microangiopathy is the fundamental pathophysiological abnormality that results in several end-organ damage in diabetes.
Adamiec, "Neutrophil surface expression of CD11b and CD62L in diabetic microangiopathy," Acta Diabetologica, vol.
14) Diabetic cheiroarthropathy, that may develop due to the changes in collagen biochemistry and diabetic microangiopathy of the skin, tends to respond well to physical therapy.
There is debate about the exact etiology of diabetic muscle infarction but several theories have been postulated such as vasculitis, thrombosis of small vessels, atherosclerotic plaquing, and diabetic microangiopathy.
Decreasing incidence of severe diabetic microangiopathy in type 1 diabetes.
In diabetic nephropathy, disturbances in the renal microcirculation may be relevant (eg, severe diabetic microangiopathy with arteriolar hyalin, possible more-prominent loss of peritubular capillaries).
Cramps and muscular pain: prevention with pycnogenol in normal subjects, venous patients, athletes, claudicants and in diabetic microangiopathy.
There is compelling evidence from randomized, controlled trials that diabetic microangiopathy and neuropathy can be reduced by tight glycemic control (2,3).
13) The accumulation of basement membrane is considered to be a characteristic of diabetic microangiopathy, as excess glucose has shown to result in the stimulation of the basement membrane accumulation.
There are data from postmortem studies of diabetic individuals to suggest that the lung is a target organ for diabetic microangiopathy, as well as indirect data showing that diabetes may contribute to lower diffusion capacity.