glomerulopathy

(redirected from diabetic glomerulopathy)

glomerulopathy

 [glo-mer″u-lop´ah-the]
any disease, especially any noninflammatory disease, of the renal glomeruli.
diabetic glomerulopathy intercapillary glomerulosclerosis.

glo·mer·u·lop·a·thy

(glō-mer'yū-lop'ă-thē),
Glomerular disease of any type.
[glomerulus + G. pathos, suffering]

glomerulopathy

/glo·mer·u·lop·a·thy/ (glo-mer″u-lop´ah-the) any disease of the renal glomeruli.
diabetic glomerulopathy  intercapillary glomerulosclerosis.

glo·mer·u·lop·a·thy

(glō-mer'yū-lop'ă-thē)
Glomerular disease of any type.
[glomerulus + G. pathos, suffering]

glomerulopathy

any disease, especially any noninflammatory disease, of the renal glomeruli.

Finnish-Landrace glomerulopathy
see membranoproliferative glomerulonephritis.
Samoyed hereditary glomerulopathy
inherited as an X-linked dominant trait it affects males more severely and earlier than females; wasting and proteinuria are characteristic; males are dead by 15 months of age.
References in periodicals archive ?
Diabetic glomerulopathy is characterized by excessive accumulation of glomerular basement membrane and mesangial matrix.
Figure 2 shows a light microscopic examination of kidneys from diabetic animals revealing diabetic glomerulopathy characterized by thickening of the glomerular basement membrane, mesangial matrix expansion, arteriolar hyalinosis, and large hyaline proteinaceous droplets (arrow) within the glomeruli (H&E, 200x), occluding capillary loops and attached outside the Bowman's capsule.
Capillaries are variably thickened as compared with the uniform thickening seen in diabetic glomerulopathy.
The diagnosis of diabetic kidney disease (DKD) can usually be made with careful clinical and laboratory assessment, but kidney biopsy might be required to prove the presence of diabetic glomerulopathy.
The key changes in diabetic glomerulopathy is thickening of the basement membrane, mesangial cell hypertrophy and accumulation of mesangial matrix (Kimmelstiel-Wilson nodules).
Structural and functional changes in diabetic glomerulopathy.
Microalbuminuria in the type 2 patient may be caused by the endothelial dysfunction associated with insulin resistance rather than diabetic glomerulopathy, which would explain the lower rate of progression to diabetic nephropathy in the type 2 patient.
Course of glomerular filtration rate in albuminuric type-2 diabetic patients with or without diabetic glomerulopathy.
Taken together, these results indicate that Cnidii rhizoma and Tabanus inhibit the high glucose-induced GMC proliferation partially through suppressing the ECM accumulation and TGF-[beta]1 production, suggesting that these medicines may be a promising agent for treating the development and progression of diabetic glomerulopathy.
For example, question 2*4 assumed a basic knowledge of histologic and ultrastructural characteristics of diabetic glomerulopathy (diabetic patient with a transplanted kidney).
In contrast, several studies report that diabetic patients without retinopathy may have diabetic glomerulopathy or nephropathy at a rate of 44 to 70%, indicating that the possibility of DN cannot be excluded confidently by the absence of diabetic retinopathy, although the absence of retinopathy strongly favors NDRD.