The formation of cilia.
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It has been revealed that SNX10 protein is involved in ciliogenesis (5).
Their topics include post-translational modifications of tubulin and cilia, the central apparatus of cilia and eukaryotic flagella, transition zone migration: a mechanism for cytoplasmic ciliogenesis and postaxonemal centriole elongation, primary cilia and mammalian hedgehog signaling, G-protein-coupled receptor signaling in cilia, ciliary mechanisms of cyst formation in polycystic kidney disease, sperm sensory signaling, cilia and ciliopathies in congenital heart disease, photoreceptor cilia and retinal ciliopathies, and the evolution of cilia.
Influence of culture duration and ciliogenesis on the relationship between ciliary beat frequency and temperature in nasal epithelial cells.
The genetic contributors to NTDs are still poorly understood, but include genes involved in the folic acid pathway, in both fetal and maternal metabolism, and genes coding for many aspects of embryological control such as planar cell polarity and ciliogenesis.
These histological factors included reductions in the number of glandular ostia, an irregular distribution of glandular ostia, incomplete ciliogenesis, and reductions in the ciliated cell ratio.
The von Hippel-Lindau tumor suppressor protein controls ciliogenesis by orienting microtubule growth.
The researchers demonstrated that HYLS1 stably incorporates into centrioles during their assembly, and plays a crucial role in the early step steps of the ciliogenesis pathway.
A role for Alstrom syndrome protein, Alms1, in kidney ciliogenesis and cellular quiescence.
tenuis at 10-13[degrees]C Event Time Fertilization 0 First polar body 30 min First cleavage 2 h 15 min Second cleavage 3 h Third cleavage 3 h 45 min Ciliogenesis 15 h Swimming blastulae 16 h Gastrulation ~24 h Episphere eyespots ~42-48 h Feeding ~70-75 h Telotroch ~16 d First accessory eyespot ~24-30 d Chaetae ~24-30 d Metamorphosis ~35 d After the onset of feeding at 70-75 h, development rate varies substantially among cultures; times recorded are for rapidly developing cultures.
Kidney-specific inactivation of the KIF3A subunit of kinesin-II inhibits renal ciliogenesis and produces polycystic kidney disease.