Protein-serine/threonine kinases, myo-inositol-1-phosphate synthases and calmodulins, as the members of ABA and IP3/Ca2+ signal transduction pathway, played an important role in the low temperature stress.
Auxin responsive and transport protein, abscisic acid receptor, serine/threonine protein kinases and calmodulin were involved in signal transduction under low temperature stress.
In our experiment, 4 myo-inositol-1-phosphate synthase genes and 3 calmodulin genes participated in the resistance to low temperature stress, and the expression of calmodulin genes continued to increase during low temperature treatment.
The downstream components; mitogen-activated protein kinases, Hsp90 and calmodulins are involved in the activation of Heat-shock transcription factors (Hsf).
Among different classes of Ca2+ sensors calmodulin (CaM), calcineurin B-like proteins (CBLs) and CBL-interacting protein kinases (CIPKs), are the best characterized (Reddy et al.