bulbospinal

bul·bo·spi·nal

(bŭl'bō-spī'năl),
Relating to the medulla oblongata and spinal cord, particularly to nerve fibers interconnecting the two.
Synonym(s): spinobulbar

bul·bo·spi·nal

(bŭl'bō-spī'năl)
Relating to the medulla oblongata and spinal cord, particularly to nerve fibers interconnecting the two.
Synonym(s): spinobulbar.

bulbospinal

nerve tracts from medulla oblongata to spinal cord
References in periodicals archive ?
So ciliary pain is referred to areas associated with cervical segments which connect with the superior cervical ganglion and the somatic outflow from which is represented by bulbospinal root of trigeminal and upper cervical nerves.
These include bilateral Bell's palsy, diabetes, Guillain-Barre syndrome, viral infections, syphilis, basal skull fractures, pregnancy, brainstem encephalitis, cryptococcal meningitis, systemic lupus erythematosus, bulbospinal muscular atrophy and borreliosis.
4,5) Specific genetic tests are available for X-linked bulbospinal neuronopathy (Kennedy's disease), which causes a slowly progressive lower motor neurone syndrome, sensory neuropathy, and partial androgen insensitivity leading to gynaecomastia and the recessive form of proximal spinal muscular atrophy which can occasionally come on in adult life.
The author was initially given two synonyms for SBMA by a physician: Kennedy disease and X-linked bulbospinal neuronopathy.
Expression of X-linked bulbospinal muscular atrophy [Kennedy disease] in two homozygous women.
bulbospinal Alexanders (type 2), which primarily has an adult onset with the presence of muscle weakness, hyperreflexia, bulbar or pseudobulbar symptoms, signal abnormalities and atrophy observed in an MRI of the medulla oblongata and upper cervical spinal cord
The fatal sequelae of this type of bite are bulbospinal paralysis, including respiratory paralysis.
Another potential contribution to the pain-generating mechanism is the role of descending bulbospinal monoaminergic pathways.
The symptoms of detrusor hyperreflexia, such as frequency, urge incontinence, and incomplete emptying, are caused by spinal cord plaques disrupting the bulbospinal pathways from the pontine micturition center (Dierich, 2000; Goodwin & Fowler, 1997; MSCCPG, 1999; Schapiro, 1998).
Giordano's recent review of pain modulatory mechanisms described the involvement of several systems, including, but not limited to, intraspinal-segmental pain modulation, bulbospinal pain modulation, centrifugal pain modulation involving midbrain and descending inhibitory controls, opioid and nonopioid hypothalamicpituitary analgesia, and corticolimbic inhibitory processing [147].