beta-adrenergic receptors


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β-ad·re·ner·gic re·cep·tors

adrenergic receptors in effector tissues capable of selective activation and blockade by drugs; conceptually derived from the ability of certain agents, such as propranolol, to block only some adrenergic receptors and of other agents, such as isoproterenol, to activate only the same adrenergic receptors. Such receptors are designated as β-receptors. Their activation results in physiologic responses such as increases in cardiac rate and force of contraction (β1), and relaxation of bronchial and vascular smooth muscle (β2) contained in skeletal muscle.

β-ad·re·ner·gic re·cep·tors

(adrĕ-nĕrjik rĕ-septŏrz)
Those in effector tissues capable of selective activation and blockade by drugs.
References in periodicals archive ?
1986) Cloning of the gene and cDNA for mammalian beta-adrenergic receptor and homology with rhodopsin.
Interactive effects between polymorphisms in the beta-adrenergic receptors and longitudinal changes in obesity.
5) At that time, it was shown that aerobic exercisers had greater alpha-adrenergic receptor density and significantly lower beta-adrenergic receptor density than weight trainers.
Beta-adrenergic receptor subtype-specific signaling in cardiac myocytes from beta(1) and beta(2) adrenoceptor knockout mice.
Beta-blocking drugs such as carvedilol reduce the heart's workload by blocking beta-adrenergic receptors on cells.
One of the enzyme's regulatory functions is to turn off beta-adrenergic receptors - molecular 'switches' that drive heart contraction.

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