basal cell hyperplasia

ba·sal cell hy·per·pla·si·a

increase in the number of cells in an epithelium resembling the basal cells.
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Because of the unusual morphology seen in many of these carcinomas as well as the p63-positive immunoprofile, the main differential diagnoses include basal cell proliferations, that is, basal cell hyperplasia and basal cell carcinoma.
Prostate lesions with cribriform or solid architectures range from benign and proliferative, such as central zone glands, clear cell cribriform hyperplasia, and basal cell hyperplasia, to invasive cribriform carcinoma.
Basal cell hyperplasia and large vacuolated epithelial cells with a clear cytoplasm are typical.
Basal cell hyperplasia in the peripheral zone of the prostate.
6,15) Epidemiologic studies suggest that there is an increased risk of invasive squamous cell carcinoma in patients with basal cell hyperplasia (relative risk, 2.
4) In a study that focused on TURP chips, cases misinterpreted as adenocarcinoma included atypical adenomatous hyperplasia (26% of the false-positive cases), basal cell hyperplasia (26%), atrophy (16%), sclerosing adenosis (10%), high-grade prostatic intraepithelial neoplasia (10%), xanthogranulomatous prostatitis (6%), florid cribriform hyperplasia (3%), and postatrophic hyperplasia (3%).
Although esophageal eosinophilia persists over time, a significant decrease in both eosinophils and basal cell hyperplasia, as well as papillary elongation, has been found.
Mucosal abnormalities that accompany squamous cell carcinoma include basal cell hyperplasia, squamous metaplasia, dysplasia, and carcinoma in situ.
Differentiation of adenoid cystic/basal cell carcinoma from basal cell hyperplasia and cribriform pattern of acinar adenocarcinoma may be difficult.
Basal cell hyperplasia, mild atypia, and numerous mitoses, some of which may appear atypical, characterize recovery from chemotherapeutic injury.
Following cryosurgery, the prostate shows typical features of repair, including marked stromal fibrosis and hyalinization, basal cell hyperplasia with ductal and acinar regeneration, squamous metaplasia, urothelial metaplasia, and stromal hemorrhage (Figure 4, A), as well as hemosiderin deposition (Figure 4, B).
In the benign cases, they were noted only in occasional nuclei in a few glands and were seen in association with inflammation, basal cell hyperplasia, Paneth cell-like metaplasia, or atrophy, as shown in Table 4.

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