antithyroid


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antithyroid

 [an″te-thi´roid]
suppressing or inhibiting thyroid activity.

an·ti·thy·roid

(an'tē-thī'royd),
Relating to an agent that suppresses thyroid function (for example, propylthiouracil).

antithyroid

/an·ti·thy·roid/ (-thi´roid) counteracting thyroid functioning, especially in its synthesis of thyroid hormones.

antithyroid

adjective Referring to an effect that counters thyroid activity.
 
noun An agent that counters thyroid activity.

antithyroid

suppressing thyroid activity.

antithyroid drugs
References in periodicals archive ?
Antithyroid peroxidase (TPO) antibodies will be elevated in more than half of patients, and antithyroglobulin antibodies in about one fourth of patients.
None of the patients used any antithyroid treatments before the hand grip strength, function, and dexterity evaluations conducted by the same investigator (S.
Although our patient with GRTH (case 1) was asymptomatic, antithyroid drug treatment had unnecessarily been recommended just based on laboratory results.
Treatment can include beta-blockers, antithyroid drugs that lower thyroid hormone concentrations, and radioactive iodine treatment that destroys the thyroid gland.
Women who have negative TRAb and do not require antithyroid drugs have a very low risk of fetal or neonatal thyroid dysfunction.
Italian specialists in endocrinology, internal medicine, and clinical and experimental medicine also address transient forms of hyper- and hypothyroidism, subclinical hyper- and hypothyroidism, aspects of thyroid disease in the elderly and pregnant women, iodine deficiency, antithyroid treatment, thyroid disruptors, and thyroid in the intensive care unit.
The underlying cause must be addressed with antithyroid medications (such as propylthiouracil or methimazole) or ablation of the thyroid.
Several recent studies, including the National Health and Nutrition Examination Survey III, suggest a much lower upper limit of normal once individuals with positive antithyroid antibodies or a family history of thyroid disease have been excluded.
The clinical presentation consists of a subacute, sometimes relapsing-remitting, steroid-responsive encephalopathy with diffuse or focal neurologic signs, headache, altered cognitive function, neuropsychiatric symptoms, diffuse electroencephalographic abnormalities and elevated levels of antithyroid antibodies.
131]I fractional uptake, homogeneity of distribution, and effective half-life); and preceding antithyroid drug therapy .
evaluated the relevant autoimmune markers in such cases_sera were assayed for antithyroid antibodies, antinuclear antibodies, C-reactive protein (CRP) and CD5 positive B cells.
Cytotoxic T lymphocyte-associated molecule-4 polymorphisms in Turkish Graves' disease patients and association with probability of remission after antithyroid therapy.