alpha-adrenergic receptors


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α-ad·re·ner·gic re·cep·tors

adrenergic receptors in effector tissues capable of selective activation and blockade by drugs; conceptually derived from the ability of certain agents, such as phenoxybenzamine, to block only some adrenergic receptors and of other agents, such as methoxamine, to activate only the same adrenergic receptors. Such receptors are designated as α-receptors. Their activation results in physiologic responses such as increased peripheral vascular resistance, mydriasis, and contraction of pilomotor muscles.

α-ad·re·ner·gic re·cep·tors

(adrĕ-nĕrjik rĕ-septŏrz)
Those in effector tissues capable of selective activation and blockade by drugs; conceptually derived from the ability of certain agents, such as phenoxybenzamine, to block only some adrenergic receptors and of other agents, such as methoxamine, to activate only the same adrenergic receptors. Such receptors are designated as α-receptors. Their activation results in physiologic responses such as increased peripheral vascular resistance, mydriasis, and contraction of pilomotor muscles.

α-adrenergic receptors

see adrenergic receptors.
References in periodicals archive ?
It showed that blocking the beta-receptor alone promotes cardiac remodelling via growth of cardiac fibroblasts induced by alpha-adrenergic receptor signaling.
This same lipolytic response is blunted by alpha-adrenergic receptors.
Mechanism of Action: ProAmatine forms an active metabolite, desglymidodrine, that is an alpha1-agonist, and exerts its actions via activation of the alpha-adrenergic receptors of the arteriolar and venous vasculature, producing an increase in vascular tone and elevation of blood pressure.

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