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activated protein C resistance

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activated protein C resistance
APC resistance Hematology A condition caused by an inherited defect in the anticoagulant response to APC and clinically characterized by ↑ venous thrombosis; it is responsible for 20-50% of DVT Pathogenesis Protein C, a key regulator of coagulation, circulates in an inactivated form and is activated by the binding of thrombin to thrombomodulin receptors on vascular endothelial cells; once activated, protein C lyses coagulation factors Va and VIIIa; APCR may be due to a selective defect in factor V coagulant function
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Reported causes of thrombophilia in HIV-infected subjects include antiphospholipid syndrome [1], increased platelet activation [1], elevated homocysteinemia [2], elevated plasma factor VII activity [2], lupus anticoagulant [3], activated protein C resistance [3], protein C deficiency [3,4], and acquired protein S deficiency [4-10].
Hypercoagulability due to protein S deficiency in HIV-seropositive ... by Orlovic, Dragana; Smego, Raymond A. / International Journal of Collaborative Research on Internal Medicine & Public Health (IJCRIMPH)
Activated protein C resistance (Factor V Leiden) and thrombosis: factor V mutations causing hypercoagulable states.
Combined heterozygote factor V Leiden mutation and anticardiolipin ... by Karadurmus, Nuri; Ozturk, Cengiz; Sonmez, Alper; Dogru, Teoman; Inal, Satilmis; Tasci, Canturk; Oztu / The Anatolian Journal of Cardiology (Anadolu Kardiyoloji Dergisi)
Patients, especially those with an underlying hypercoagulable state such as activated protein C resistance, malignancy, pregnancy, and antiphospholipid syndrome, are at risk of developing deep vein thrombosis from blood stasis from prolonged immobilization.
March madness-associated deep vein thrombosis by Roy, Michael J. / Southern Medical Journal
 
 
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