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hyperphosphatemia

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hyperphosphatemia

 [hi″per-fos″fah-te´me-ah]
an excess of phosphates in the blood.
Miller-Keane Encyclopedia and Dictionary of Medicine, Nursing, and Allied Health, Seventh Edition. © 2003 by Saunders, an imprint of Elsevier, Inc. All rights reserved.

hy·per·phos·pha·te·mi·a

(hī'per-fos-fă-tē'mē-ă),
Abnormally high concentration of phosphates in the circulating blood.
Farlex Partner Medical Dictionary © Farlex 2012

hyperphosphatemia

(hī′pər-fŏs′fə-tē′mē-ə)
n.
An abnormally high concentration of phosphates in the blood.
The American Heritage® Medical Dictionary Copyright © 2007, 2004 by Houghton Mifflin Company. Published by Houghton Mifflin Company. All rights reserved.

hyperphosphatemia

Nephrology↑ Phosphate > 1.5 mmol/L–US: 4.5 mg/dL in blood Etiology ↑ hGH, either physiologic with growth spurts or pathologic in gigantism and acromegaly, ↓ PTH, pseudohypoparathyroidism or in renal failure
McGraw-Hill Concise Dictionary of Modern Medicine. © 2002 by The McGraw-Hill Companies, Inc.

hy·per·phos·pha·te·mi·a

(hī'pĕr-fos'fă-tē'mē-ă)
Elevation of phosphorus concentration in blood; may be due to consumption of large amounts of phosphorus-rich foods and drinks or to renal insufficiency. It may also be caused by hypocalcemia, hyperparathyroidism, or overuse of phosphate-containing laxatives or antacids. With concurrent hypocalcemia, tetany and seizures may occur.
Synonym(s): hyperphosphataemia.
Medical Dictionary for the Health Professions and Nursing © Farlex 2012

hy·per·phos·pha·te·mi·a

(hī'pĕr-fos'fă-tē'mē-ă)
Abnormally high concentration of phosphates in the circulating blood.
Synonym(s): hyperphosphataemia.
Medical Dictionary for the Dental Professions © Farlex 2012
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References in periodicals archive
* Ardelyx Inc (NASDAQ: ARDX) (reported positive results from a late-stage of its lead compound tenapanor in combination with phosphate binders to treat hyperphosphatemia in patients with chronic kidney disease on dialysis)
Ardelyx reported results from AMPLIFY, a pivotal Phase 3 study of tenapanor in combination with phosphate binders in patients with chronic kidney disease, or CKD, on dialysis whose hyperphosphatemia was not previously controlled with binders alone.
KEYWORDS: Tumor lysis syndrome, hyperuricemia, hyperphosphatemia, hypocalcemia, acute kidney injury.
Iron Overload: Increases in serum ferritin and transferrin saturation (TSAT) were observed in clinical trials with AURYXIA in patients with chronic kidney disease (CKD) on dialysis treated for hyperphosphatemia, which may lead to excessive elevations in iron stores.
Auryxia is the only oral iron tablet approved in the US to treat non-dialysis-dependent-chronic kidney disease adult patients for iron deficiency anemia, and dialysis-dependent CKD adult patients for hyperphosphatemia.
Sodium phosphate enemas and some rodenticides can also cause hyperphosphatemia. Hyperthyroidism in cats may cause an increase in phosphate, as thyroxin (thyroid hormone) can interfere with renal clearance of phosphate.
Patients with HFTC usually present with hyperphosphatemia and tumor-like soft tissue calcifications.
M2 EQUITYBITES-February 8, 2019-Ardelyx Inc begins Phase 2 clinical study of tenapanor for the treatment of hyperphosphatemia dialysis patients in Japan
Global Banking News-February 8, 2019-Ardelyx Inc begins Phase 2 clinical study of tenapanor for the treatment of hyperphosphatemia dialysis patients in Japan
Keryx's Auryxia (ferric citrate) is a US Food and Drug Administration -approved medicine to treat dialysis dependent CKD patients for hyperphosphatemia and non-dialysis dependent CKD patients for iron deficiency anemia.
According to the K/DOQI guidelines, we considered hypocalcemia as a total serum calcium level of <8.4 mg/dl, hyperphosphatemia as a serum phosphorus level of >5.5 mg/L, and high iPTH as a serum iPTH level of >300 pg/ml.[5] Associated parameters were compared between the two groups, and then overall data in Hebei province were compared to those reported in DOPPS4[6],[7] and Chinese DOPPS.[8]
PHP is a rare condition characterized by a biochemical pattern of hypocalcemia, hyperphosphatemia, and increased PTH concentrations (2).
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