Comparison of Clinical Features with Reference to Haematoiogicai Values Signs Hb (gm/dl) Lymphadenopathy (n = 7) 6.36 [+ or -] 0.43
Hepatomegaly (n = 16) 6.03 [+ or -] 1.23 Splenomegaly (n = 21) 4.88 [+ or -] 2.14 p-Value 0.52 Signs TLC (/ul) Lymphadenopathy (n = 7) 69246.799 [+ or -] 33672.368
Hepatomegaly (n = 16) 63226.735 [+ or -] 30276.269 Splenomegaly (n = 21) 47874.394 [+ or -] 2163.351 p-Value 0.54 Signs Platelet Count (Lacs/cmm) Lymphadenopathy (n = 7) 130200.565 [+ or -] 8349.952
Hepatomegaly (n = 16) 108775.755 [+ or -] 51687.363 Splenomegaly (n = 21) 103181.282 [+ or -] 32269.779 p-Value 0.51
Comparing the criteria, it is obvious that the majority of our patients satisfied the criteria from 2005, which include clinical manifestations such as
hepatomegaly and hemorrhages.
Hepatomegaly is observed with a lower frequency compared with splenomegaly (2).
This patient presented with massive ascites and
hepatomegaly, severe malnutrition resulting in growth retardation, short stature, muscle wasting, fixed flexion deformity and radiologically confirmed fracture of left humerus.
Given this rise in liver enzymes associated with abdominal pain and
hepatomegaly in the setting of type 1 diabetes mellitus, we initiated the workup to evaluate the etiology behind this presentation.
No
hepatomegaly and no growth delay were noticed; the patient weighed 16 kg (z score of 0.9) and measured 95.5 cm (z score of -0.35), with a BMI of 17.8 kg/m2 (z score of 1.63).
Significant jugular venous distension and
hepatomegaly were present on physical examination.
None of the patient in the category of DF had bleeding manifestations, jaundice, lymphadenopathy, splenomegaly, pleural effusion or ascites; however 5 of them had
hepatomegaly.
Therefore in adults with fever, jaundice,
hepatomegaly and altered liver function tests, the diagnosis of dengue infection should be strongly considered in areas where dengue infection is endemic.
The patient has posterior cervical lymphadenopathy but no
hepatomegaly or splenomegaly.
Congenital generalized lipodystrophy (CGL) is a rare autosomal recessive disorder characterized by generalized absence of adipose tissue, extreme insulin resistance, hypertriglyceridemia,
hepatomegaly hepatic steatosis, and early onset of diabetes.
A CT scan of the abdomen and pelvis with and without contrast showed bilaterally enlarged calcified adrenal glands,
hepatomegaly with fatty change, splenomegaly, and mild ascites (Figures 4-6).