KeyWords:
Adenoid hypertrophy, Nasal obstruction, Frequency.
(8) also divided patients according to airway obstruction caused by
adenoid hypertrophy into three groups; less than 50%, 50% to 75%, and more than 75%; only two patients out of 34 were in the first group.
Montelukast in
adenoid hypertrophy: its effect on size and symptoms.
Chronic rhinosinusitis and
adenoid hypertrophy in children.
(1,7,14) Moreover, the statement that children with AR have a higher tendency than children with NAR to suffer from obstructive adenoids has been based on the assumption that inhaled allergens--such as molds, house dust mites, or even food allergens--are transported to immunocompetent nasopharyngeal cells and thus may promote
adenoid hypertrophy. (1,4,15) Although the gold standard test for detection of hypersensitivity to allergens is the skin prick test, nasal smear eosinophilia has been shown to be of high sensitivity in the diagnosis of AR.
Age, year 9 [+ or -] 4(3-20) (c) Gender, % Male 53 Ethnic group, % White 25 Mulatto 74 Black 2 Body mass index, kg/[m.sup.2] 16 [+ or -] 2(11-22) (c) Shwachman score 85 (65-100) (d) Endoscopic exam, % Middle meatus purulent secretion 38
Adenoid hypertrophy 18 Polyps 12 CT scan, % Maxillary opacification (a) 73 Ethmoid opacification (a) 61 Ostiomeatal complex opacification (b) 61 Note.
Adenoid hypertrophy is not present in the endoscopic nasopharyngeal examination.
Recurrent infections, including recurring otitis media (ROM) and
adenoid hypertrophy (AH), are common indications for adenoidectomy (3).
However, an adenoid X-ray examination showed posterior nasopharyngeal soft tissue that was indenting and narrowing the nasopharyngeal air column (Figure 1), which suggested
adenoid hypertrophy. We observed a faint soft tissue structure (19 mm x 13 mm) in the oropharynx.
Nasal obstruction may be the cardinal presenting symptom of many common disease processes, such as rhinitis, sinusitis, septal deviation,
adenoid hypertrophy, and nasal trauma13-15.
In persistent OME with hearing impairment, the current standard of care is conservative treatment and if necessary insertion of tympanostomy tube as the first step followed by adenoidectomy in resistant cases or when symptoms of
adenoid hypertrophy are prominent.