Intercellular adhesion molecule-1 (ICAM-1,
CD54) is increased in adhesive capsulitis.
Evaluation of the phenotypic characterization of MSC, derived from bone marrow tissue, indicated the expression of CD90 (clone 0x-75 mouse), CD73 (clone 5 F/ B9 mouse), and
CD54 (clone 1A29 mouse) in 86.8%, 94.0%, and 95.1% of the cells, respectively, while CD45 was not expressed in 97.0% of the cells confirming its classification.
DC in MDS express lower levels of CD1a,
CD54, CD80, and MHC II molecules [26].
In addition, phenotypic characterisation of AMs and IMs in mice revealed differences in the expression levels of MHC classII, CD11b, CD14, CD45,
CD54, CD68, CD71, CD204, CD206, and Siglec-F [5-9].
When incubated with VEGF, the expression of VEGFR1, VEGFR2, von Willebrand factor, and
CD54 is increased [104, 105].
Once prepared, the platelet-poor plasma is labeled with fluorescent monoclonal antibodies for staining specific surface antigens expressed on endothelial cells such as CD31 and
CD54, and the cytometric analysis is performed by applying forward and side light scatter with calibration beads of a known diameter [58].
Furthermore, they demonstrated that statins reduce the cell surface expression of other immunoregulatory molecules, such as MHC-I, CD3, CD4, CD8, CD28, CD40, CD80, CD86, and
CD54 (Kuipers et al).
Finally, lack of CD29 ([beta]-1 integrin) and
CD54 (ICAM-1) in IVLBCL maybe potentially useful diagnostic feature.