Virchow-Robin spaces


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Virchow-Robin spaces

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EPVS, also known as Virchow-Robin spaces, are most likely to be identified in T2-weighted MRI and are characterized by punctate or linear signal intensities similar to cerebrospinal fluids.
Risk factors of dilated Virchow-Robin spaces are different in various brain regions.
8,9) However dilatation of Virchow-Robin spaces tend to be the common finding and should raise concern for cryptococcal infection in any immunocompromised patient.
Areas smaller than 2 mm in diameter, present in the lower third of corpus striatum of the basal ganglia and demonstrating isointensity to cerebrospinal fluid, differentiate normally dilated Virchow-Robin spaces from lacunar infarcts [45].
The second is that leukemic cells passing through the first dura, and then the subarachnoid and Virchow-Robin spaces invade the brain surface (17).
Hypothesized structural changes in the brain after acute or chronic exposure to HA or hypobaric hypoxia, by various authors include peri-ventricular damage, fronto-temporal changes, cortical atrophy and Virchow-Robin spaces enlargement in up to 92% of the HA climbers with an increased risk in amateurs versus professional climbers4,5.
There were perivascular inflammatory infiltrates with occasional hemosiderin laden macrophages extending into the Virchow-Robin spaces.
NMO-IgG positive sera show a typical staining pattern around blood vessels, along the pial surfaces and Virchow-Robin spaces (see Figure 2A for a cartoon representation of this method and Figure 3A for an example).
AQP-4 is the main water channel in the CNS where it is expressed around cerebral microvessels, pia mater and Virchow-Robin spaces
One possible mechanism is that leukemic cells from the bone marrow of the skull travel to the adjacent dura, and then to the subarachnoid space via the Virchow-Robin spaces (VRS; Woo et al.
On microscopic examination, all sections of the brain showed infiltration of the arachnoid and many penetrating Virchow-Robin spaces by spindle cells with dark red pigment (Fig.
In the more active cases, chronic inflammatory cells fill the Virchow-Robin spaces and extend into the neuropil, forming microscopic clusters or larger aggregates.