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pressure ulcer

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Pressure ulcer
Also known as a decubitus ulcer, pressure ulcers are open wounds that form whenever prolonged pressure is applied to skin covering bony outcrops of the body. Patients who are bedridden are at risk of developing pressure ulcers. Pressure ulcers are commonly known as bedsores.
Mentioned in: Debridement

pressure ulcer,
an inflammation, sore, or ulcer in the skin over a bony prominence, most frequently on the sacrum, elbows, heels, outer ankles, inner knees, hips, shoulder blades, and occipital bone of high-risk patients, especially those who are obese, elderly, or suffering from chronic diseases, infections, injuries, or a poor nutritional state. It results from ischemic hypoxia of the tissues caused by prolonged pressure on them. Pressure ulcers are most often seen in aged, debilitated, immobilized, or cachectic patients. The sores are graded by stages of severity. Prevention of pressure ulcers is a cardinal aspect of nursing care. Treatment specific to the location and the extent of the condition is planned. Also called bedsore, decubitus ulcer, pressure necrosis, pressure sore.

pressure ulcer 
an ulcer due to local interference with circulation; persons most at risk are those who are emaciated (nutritionally deficient in protein), obese, immobilized by traction or anything else, diabetic, or suffering from a circulatory disorder. Because urine and feces contribute to skin breakdown, incontinent patients are at high risk. Absence of sensation, advanced age, and immunodeficiency are also risk factors. Called also decubitus ulcer and, popularly, bedsore or pressure sore.

Three major factors in the development of pressure ulcers are (1) prolonged pressure on a part due to the weight of the body or a limb; (2) a shearing force that exerts downward and forward pressure on tissues beneath the skin (this can occur when the patient slides downward while sitting in a bed or chair, or when bedclothes are forcibly pulled from under the patient); and (3) nutritional status: good nutrition is essential for preventing pressure ulcers and healing already existing ones; a dietary deficiency should be suspected with a loss of 5 per cent or more of body weight or a serum albumin level below 3.5 mg/dl.

A pressure ulcer usually occurs over a bony prominence at the sacrum, hip (trochanter), heel, shoulder, or elbow. The lesion begins as a reddened area, which can quickly involve deeper structures and become an ulcer.
Prevention. Repositioning the patient must be done as often as necessary to prevent impairment of circulation to any one part. For some patients this may mean turning and repositioning every hour. Gentle massage of the area stimulates circulation to the areas most likely to be affected, but reddened areas should never be massaged, because massage encourages tissue breakdown. Thorough cleansing, especially to remove perspiration, urine, and feces, helps prevent chemical breakdown of the skin and aids in the removal of bacteria. Numerous different pressure-relieving devices are available.
Stages. For purposes of assessment, treatment, and evaluation of effectiveness of nursing and medical intervention, the pathologic changes occurring in the development of a pressure ulcer are divided into four stages.

In Stage 1 the area of skin is deep pink, red, or mottled. Digital pressure on the area will cause temporary blanching for up to 15 minutes after pressure is released. The skin will feel very warm and firm or tightly stretched across the area. At this stage no destruction of tissue has occurred and the condition is reversible. It is essential that the area be relieved of prolonged pressure, and that shearing forces be avoided. The reddened area may need protection by covering it with either a transparent film or a skin barrier.

Stage 2 is characterized by blistering, cracking, or abrasion of the skin. The surrounding skin is reddened and probably will feel hot or warmer than normal. Since there is now an opening in the skin for the entrance of bacteria, treatment must include cleaning the wound and providing some type of dressing or cover in addition to relieving pressure on the area.

Stage 3 is characterized by a craterlike sore with involvement of the underlying structures. Bacterial infection is almost always present at this stage and accounts for continued erosion of the ulcer and the production of drainage. Irrigation of the wound usually is done each time the dressing is changed. Wound débridement may be necessary to promote healing.

At Stage 4 there is deep ulceration and necrosis involving underlying muscle and possibly bone tissue. At this point the ulcer usually is extensively infected. It can be dry, black in color, and covered with a tough accumulation of necrotic tissue, or it can be wet and oozing dead cells and purulent exudate. Deep and extensive ulceration and tissue loss may require surgical repair with myocutaneous flaps to close the defect.

Topical applications vary widely. The diligence with which the prescribed regimen is carried out greatly influences its effectiveness. Guidelines summarizing current recommended practice for the treatment of pressure ulcers (publication number 95-0653) are available from the Agency for Health Care Policy and Research, P.O. Box 8547, Silver Spring, MD 20907-8547 (telephone 800-358-9295).
Pressure areas in common resting positions: A, Fowler's; B, supine; C, prone; and D, side-lying.

pressure ulcer
Bedsore A decubitus ulcer on dependent sites, usually lumbosacral, but also on heels, knees, or vertebrae, which is most common in the bed-ridden elderly, seen in up to14 of nursing home residents, and associated with an ↑ mortality Risk factors Nonblanchable erythema, lymphopenia, immobility, dry skin, ↓ body weight, activity limited to bed or chair Management A 'cocktail' of recombinant PDGF, proteases, cell-adhesion molecules may induce healing of recalcitrant PUs
Pressure ulcer
Stage I Nonresolving erythema with no break in skin
Stage II Erythema with superficial disruption of skin, abrasions, vesiculation
Stage III Full-thickness loss of skin with serosanguineous drainage
Stage IV Full-thickness loss of skin and invasion of deeper tissue

Patient discussion about pressure ulcer.

Q. I ask a client's Dr. to script flexaril for a lower back spasm and he made it for a drug called zanaflex? I am unfamiliar with zanaflex, what is the difference between it and flexaril 25mg? Benefits? Risks? I got him to order the air mattress and extended bed because client is 6'3" and is already bedridden on my 1st day..try to beat the skin breakdown, already stage I decubitis ulcers. I tried to talk the client into slideboard and lift away arm wheelchair...noway..he wants to walk bent with a rolling walker. He already had a lift chair delivered, so he just goes from bed to lift chair. He refuses to let me bathe him. He can't see, and he has me check his draw up on insulin to make sure it's right. He sends the P.T. man right back out the door after he signs the sheet. Difficult pt.!

A. Flexeril and Zanaflex are different drugs but are both muscle relaxants. There are hardly any differences between the two, clinically wise. If the doctor thought one is better than the other for your client I would suggest you take his advice and use the one he gave you.

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The Wound, Ostomy and Continence Nurses (WOCN) Society recognizes and supports the fact, that a pressure ulcer evaluation represents one aspect of a comprehensive patient assessment which includes, but may not be limited to: history and physical examination, risk factors for pressure ulcer development, comorbidities, individual goals, and expectations.
2) During her inpatient stay, the patient developed septicaemia from an infected pressure ulcer and subsequently died.
A pressure ulcer is a lesion caused by unrelieved pressure that causes damage to underlying tissue and a breakdown in the skin.
 
 
 
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