Accelerated Atherosclerosis

(redirected from Premature Atherosclerosis)

Accelerated Atherosclerosis

Atherosclerosis which occurs in a person with certain risk factors—e.g., SLE, diabetes, smoking, hypertension, hypercholesterolaemia, family history of early heart disease—at an earlier age than would occur in another person without those risk factors.
References in periodicals archive ?
Unrecoverable risk factors include older age, male gender, and family history of premature atherosclerosis.
Premature atherosclerosis may be the first clinical manifestation of APS, and in a case such as this where age-accelerated atherosclerosis is seen with superimposed thrombosis and subsequent rethrombosis, a hypercoagulable state such as APS should be considered.
Formation of plaques in the heart may lead to premature atherosclerosis and coronary artery disease (CAD), while formation of plaques in vessels of the brains can lead to cerebrovascular disease.
Vascular dysfunction caused by endothelial dysfunction is an early abnormality in the MetS that may contribute to premature atherosclerosis (2).
Results of this proposal will improve our understanding of the mechanisms involved in the development of premature atherosclerosis and cardiovascular complications in patients with SLE.
Research has shown that visceral fat is a source of inflammation that contributes to premature atherosclerosis and risk of acute coronary syndrome.
Interferon-alpha promotes abnormal vasculogenesis in lupus: a potential pathway for premature atherosclerosis.
1) If it is left untreated, markedly premature atherosclerosis is the usual outcome.
Irbesartan, an angiotensin type 1 receptor inhibitor, regulates markers of inflammation in patients with premature atherosclerosis.
These can hasten the development of premature atherosclerosis and coronary artery disease.
Thus, susceptibility of a human individual to premature atherosclerosis can be evaluated by: (a) obtaining a sample of DNA from the individual; and (b) evaluating the sample of DNA for the presence of nucleotides encoding a serine residue as amino acid 291 of the lipoprotein lipase gene product.
There was no history of drug abuse, past history of thromboembolism or family history of premature atherosclerosis.