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nociceptor
(redirected from Nociceptive neuron)

    Also found in: Dictionary/thesaurus, Encyclopedia, Wikipedia 0.01 sec.
nociceptor /no·ci·cep·tor/ (-sep´ter) a receptor for pain caused by injury, physical or chemical, to body tissues.nocicep´tive
no·ci·cep·tor (ns-sptr)
n.
A sensory receptor that responds to pain.
Nociceptor
A nerve cell that is capable of sensing pain and transmitting a pain signal.
Mentioned in: Migraine Headache, Pain, Pain Management
nociceptor
[nō′sēsep′tər]
a somatic and visceral free nerve ending of thinly myelinated and unmyelinated fibers. It usually reacts to tissue injury but also may be excited by endogenous chemical substances.
nociceptor [no″se-sep´tor]
a receptor for pain, stimulated by various kinds of tissue injury. adj., adj nocicep´tive.
nociceptor (nō´sisep´tr),
n somatic and visceral free nerve endings of thinly myelinated and unmyelinated fibers. They usually react to tissue injury but also may be excited by endogenous chemical substances.
nociceptor
a receptor that is stimulated by injury; a receptor for pain.
nociceptor
Pain receptor Neurology Any of a class of periarticular and mucocutaneous sense organs and neural receptors–eg, reflex loops for reception and response to pain; located primarily in the skin or viscera, nociceptors respond to chemical, mechanical, or other stimuli
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Conventional wisdom was that the nociceptive neurons in the skin can't tell the difference between heat and mechanical pain, like a pin prick.
How the body differentiates between a scorch and a scratch by Asian News International
Nociceptive processing after SCI is apparently more complicated; inhibition of below-level spinal nociceptive neurons by 5-HT has been found to be reversed by both 5-HT1A and 5-HT3 antagonists [57], but spinal 5-HT3 receptors have also been shown to facilitate at-level allodynia after SCI [58-59].
Restorative effects of stimulating medullary raphe after spinal cord ... by Hentall, Ian D.; Burns, Scott B. / Journal of Rehabilitation Research & Development
However, it was known that constriction induced by acetic acid was considered to be a non-selective anti-nociceptive model, since acetic acid indirectly induced the release of endogenous mediators stimulated the nociceptive neurons that were sensitive to non-steroidal antiinflammatory drugs (NSAIDs) (Sanchez-Mateo et al.
Antinociceptive activity of petroleum ether fraction from the MeOH ... by Chen, Yu-Feng; Li, Nan; Jiao, Yu-Liang; Wei, Peng; Zhang, Qiao-Yan; Rahman, Khalid; Zheng, Han-Chen; / Phytomedicine: International Journal of Phytotherapy & Phytopharmacology
 
 
 
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