infection interference with Mycobacterium bovis diagnostics: Natural infection cases and a pilot experimental infection.
Rapidly growing mycobacterial (RGM) species form mature colonies on culture media within seven days and include Mycobacterium fortuitum
, chelonae, and abscessus.
Disk diffusion testing with polymyxin and amikacin for differentiation of Mycobacterium fortuitum
and Mycobacterium chelonae.
5 >40 Mycobacterium tuberculosis Mycobacterium avium >40 Mycobacterium fortuitum
>40 Mycobacterium smegmatis >40 Mycobacterium intracellulare >40 Mycobacterium chimera >40 Mycobacterium nonchromogenicum >40 Mycobacterium colombiense >40 Mycobacterium abscessus subspboletti >40 Other dermatological diseases (n = 10) Pemphigus foliaceus (n = 2) >40 Pemphigus vulgaris (n = 1) >40 Nummular eczema (n = 1) >40 Lichenified eczema (n = 1) >40 Porphyria (n = 2) >40 Lupus erythematosus (n = 3) >40 Healthy individuals (n = 10) Normal skin >40 qPCR, quantitative PCR; [C.
Mycobacterium intracellulare was identified in two patients, Mycobacterium abscessus/chelonae complex was also identified in two patients, Mycobacterium fortuitum
and Mycobacterium kansasii were identified in one patient.
Among them, the rapidly growing organisms such as Mycobacterium chelonae, Mycobacterium fortuitum
and Mycobacterium abscessus are widespread in nature and in hospital environments .
Widespread primary cutaneous infection with mycobacterium fortuitum
Furthermore, bee venom displayed a significant anti-mycobacterial activity against RGM especially Mycobacterium fortuitum
(MIC 100 [micro]g/ml).
Los controles empleados para el estudio fueron cepas de referencia de Mycobacterium tuberculosis H37Rv ATCC27294 como control positivo, Mycobacterium fortuitum
ATCC6841 como control negativo y agua destilada esteril como control de reactivo.
Two weeks later, the final exit-site culture results from two different samples grew Mycobacterium fortuitum
Granulation tissue obtained from both ears yielded rapidly growing Mycobacterium abscessus and Mycobacterium fortuitum
that were resistant to trimethoprimsulfamethoxazole but sensitive to imipenem, clarithromycin, and azithromycin.
Approximately 80% of disease in humans due to RGM is caused by the nonpigmented Mycobacterium chelonae, Mycobacterium abscessus, and Mycobacterium fortuitum
, with the remainder caused by RGM of the pigmented Mycobacterium smegmatis group and other rarely pathogenic groups (1) (Table).