mutant

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Related to Mutant phenotype: back mutation

mutant

 [mu´tant]
1. in genetics, a variation that breeds true, owing to genetic changes.
2. produced by mutation.

mu·tant

(myū'tant),
1. A phenotype in which a mutation is manifested.
2. A gene that is rare and usually harmful, in contrast to a wild-type gene, not necessarily generated recently.

mutant

/mu·tant/ (mūt´'nt)
1. an organism that has undergone genetic mutation.
2. produced by mutation.

mutant

(myo͞ot′nt)
n.
An organism, cell, virus, or gene resulting from genetic mutation.
adj.
Resulting from genetic mutation: a mutant strain of bacteria.

mutant

[myo̅o̅′tənt]
Etymology: L, mutare, to change
1 n, any individual or organism with genetic material that has undergone mutation.
2 adj, relating to or produced by mutation.

mu·tant

(myū'tănt)
1. A phenotype in which a mutation is manifested.
2. A gene that is rare and usually harmful, in contrast to a wild-type gene, not necessarily generated recently.

mutant

Any organism or cell with a gene or genes that have suffered a MUTATION.

mutant

  1. any gene that has undergone a MUTATION.
  2. an individual showing the effects of a mutation, with a phenotype that is not WILD TYPE.

mu·tant

(myū'tănt)
A phenotype in which a mutation is manifested.

mutant,

n an individual showing a mutation.

mutant

1. an organism with a mutant gene which expresses itself in the phenotype.
2. a normal organism that is genetically different in one or more characteristics from an arbitrarily defined, previously existing 'wild-type' organism.
3. produced by a change in the nucleotide sequence of the genome.

conditional lethal mutant
mutations that occur in viruses, rendering them unable to grow under certain conditions which can be controlled experimentally. See permissive temperature.
defective interfering mutant
ts mutant
References in periodicals archive ?
Pisum sativum inflorescence architecture genes Gene Mutant phenotype VEGI No flowers, no [I.
This means that the resident value corresponding to the intersection point can be invaded by all mutant phenotypes.
The first involves the mutant phenotypes vestigial (vg) wing and ebony (e) body, the second the traits apterous (ap) wing and sepia (se) eye color, and the third vestigial wing and sepia eye.
Third, the biological basis for the Drosophila mutant phenotypes moves from the abstract to the tangible, in that a connection between a mutant protein and a physical trait is established.
These alternative hypotheses imply that the correlation of invertase activity with the mutant phenotype occurs because invertase activity is a consequence, not a cause of starch accumulation.
In such cases, we can replace GFP with KillerRed and rescue mutant phenotypes.
This is very inconvenient for analyzing, for example, embryonic lethal strains because the developmental stages at which the mutant phenotypes of interest and the developmental arrest by the balancer aneuploidy occur are very close together.
The mutant phenotypes proved to be stable in the open ground since nitrogenase activity was absent or remained negligible.