Dermatitis is a general term used to describe inflammation of the skin.
Most types of dermatitis are characterized by an itchy pink or red rash.
is an allergic reaction to something that irritates the skin and is manifested by one or more lines of red, swollen, blistered skin that may itch or seep. It usually appears within 48 hours after touching or brushing against a substance to which the skin is sensitive. The condition is more common in adults than in children.
Contact dermatitis can occur on any part of the body, but it usually affects the hands, feet, and groin. Contact dermatitis usually does not spread from one person to another, nor does it spread beyond the area exposed to the irritant unless affected skin comes into contact with another part of the body. However, in the case of some irritants, such as poison ivy, contact dermatitis can be passed to another person or to another part of the body.
Stasis dermatitis is characterized by scaly, greasy looking skin on the lower legs and around the ankles. Stasis dermatitis is most apt to affect the inner side of the calf.
Nummular dermatitis, which is also called nummular eczematous dermatitis or nummular eczema, generally affects the hands, arms, legs, and buttocks of men and women older than 55 years of age. This stubborn inflamed rash forms circular, sometimes itchy, patches and is characterized by flares and periods of inactivity.
is characterized by itching
, scaling, swelling, and sometimes blistering. In early childhood it is called infantile eczema and is characterized by redness, oozing, and crusting. It is usually found on the face, inside the elbows, and behind the knees.
may be dry or moist and is characterized by greasy scales and yellowish crusts on the scalp, eyelids, face, external surfaces of the ears, underarms, breasts, and groin. In infants it is called "cradle cap."
Causes and symptoms
Allergic reactions are genetically determined, and different substances cause contact dermatitis to develop in different people. A reaction to resin produced by poison ivy, poison oak, or poison sumac is the most common source of symptoms. It is, in fact, the most common allergy in this country, affecting one of every two people in the United States.
Flowers, herbs, and vegetables can also affect the skin of some people. Burns
increase the risk of dermatitis developing, and chemical irritants that can cause the condition include:
- detergents and soaps
- fabric softeners
- glues used on artificial nails
- topical medications
Contact dermatitis can develop when the first contact occurs or after years of use or exposure.
Stasis dermatitis, a consequence of poor circulation, occurs when leg veins can no longer return blood to the heart as efficiently as they once did. When that happens, fluid collects in the lower legs and causes them to swell. Stasis dermatitis can also result in a rash that can break down into sores known as stasis ulcers.
The cause of nummular dermatitis is not known, but it usually occurs in cold weather and is most common in people who have dry skin. Hot weather and stress can aggravate this condition, as can the following:
- fabric softeners
- soaps and detergents
- wool clothing
- bathing more than once a day
Atopic dermatitis can be caused by allergies, asthma
, or stress, and there seems to be a genetic predisposition for atopic conditions. It is sometimes caused by an allergy to nickel in jewelry.
Seborrheic dermatitis (for which there may also be a genetic predisposition) is usually caused by overproduction of the oil glands. In adults it can be associated with diabetes mellitus
or gold allergy. In infants and adults it may be caused by a biotin deficiency.
The diagnosis of dermatitis is made on the basis of how the rash looks and its location. The doctor may scrape off a small piece of affected skin for microscopic examination or direct the patient to discontinue use of any potential irritant that has recently come into contact with the affected area. Two weeks after the rash disappears, the patient may resume use of the substances, one at a time, until the condition recurs. Eliminating the substance most recently added should eliminate the irritation.
If the origin of the irritation has still not been identified, a dermatologist may perform one or more patch tests. This involves dabbing a small amount of a suspected irritant onto skin on the patient's back. If no irritation develops within a few days, another patch test is performed. The process continues until the patient experiences an allergic reaction at the spot where the irritant was applied.
Treating contact dermatitis begins with eliminating or avoiding the source of irritation. Prescription or over-the-counter corticosteroid creams can lessen inflammation and relieve irritation. Creams, lotions, or ointments not specifically formulated for dermatitis can intensify the irritation. Oral antihistamines
are sometimes recommended to alleviate itching, and antibiotics
are prescribed if the rash becomes infected. Medications taken by mouth to relieve symptoms of dermatitis can make skin red and scaly and cause hair loss.
Patients who have a history of dermatitis should remove their rings before washing their hands. They should use bath oils or glycerine-based soaps and bathe in lukewarm saltwater.
Patting rather than rubbing the skin after bathing and thoroughly massaging lubricating lotion or nonprescription cortisone creams into still-damp skin can soothe red, irritated nummular dermatitis. Highly concentrated cortisone preparations should not be applied to the face, armpits, groin, or rectal area. Periodic medical monitoring is necessary to detect side effects in patients who use such preparations on rashes
covering large areas of the body.
Coal-tar salves can help relieve symptoms of nummular dermatitis that have not responded to other treatments, but these ointments have an unpleasant odor and stain clothing.
Patients who have stasis dermatitis should elevate their legs as often as possible and sleep with a pillow between the lower legs.
Tar or zinc paste may also be used to treat stasis dermatitis. Because these compounds must remain in contact with the rash for as long as two weeks, the paste and bandages must be applied by a nurse or a doctor.
Coal-tar shampoos may be used for seborrheic dermatitis that occurs on the scalp. Sun exposure after the use of these shampoos should be avoided because the risk of sunburn of the scalp is increased.
Some herbal therapies can be useful for skin conditions. Among the herbs most often recommended are:
- Burdock root (Arctium lappa)
- Calendula (Calendula officinalis) ointment
- Chamomile (Matricaria recutita) ointment
- Cleavers (Galium ssp.)
- Evening primrose oil (Oenothera biennis)
- Nettles (Urtica dioica)
Contact dermatitis can be treated botanically and homeopathically. Grindelia (Grindelia spp.) and sassafras (Sassafras albidum) can help when applied topically. Determining the source of the problem and eliminating it is essential. Oatmeal baths are very helpful in relieving the itch. Bentonite clay packs or any mud pack draws the fluid out and helps dry up the lesions. Cortisone creams are not recommended.
Stasis dermatitis should be treated by a trained practitioner. This condition responds well to topical herbal therapies, however, the cause must also be addressed. Selenium-based shampoos, topical applications of flax oil and/or olive oil, and biotin supplementation are among the therapies recommended for seborrheic dermatitis.
Dermatitis is often chronic, but symptoms can generally be controlled.
Contact dermatitis can be prevented by avoiding the source of irritation. If the irritant cannot be avoided completely, the patient should wear gloves and other protective clothing whenever exposure is likely to occur.
Immediately washing the exposed area with soap and water can stem allergic reactions to poison ivy, poison oak, or poison sumac, but because soaps can dry the skin, patients susceptible to dermatitis should use them only on the face, feet, genitals, and underarms.
Clothing should be loose fitting and 100% cotton. New clothing should be washed in dye-free, unscented detergent before being worn.
Injury to the lower leg can cause stasis dermatitis to ulcerate (form open sores). If stasis ulcers develop, a doctor should be notified immediately.
and other relaxation techniques may help prevent atopic dermatitis caused by stress
Avoidance of sweating may aid in preventing seborrheic dermatitis.
A patient who has dermatitis should also notify a doctor if any of the following occurs:
- fever develops
- skin oozes or other signs of infection appear
- symptoms do not begin to subside after seven days' treatment
- he/she comes into contact with someone who has a wart, cold sore, or other viral skin infection
"Allergic Contact Dermatitis." The Skin Site. April 10, 1998 (January 11, 2006). http://www.skinsite.com/info_allergic.htm.
— An inappropriate or exaggerated genetically determined reaction to a chemical that occurs only on the second or subsequent exposures to the offending agent, after the first contact has sensitized the body.
— A group of synthetic hormones that are used to prevent or reduce inflammation. Toxic effects may result from rapid withdrawal after prolonged use or from continued use of large doses.
— A skin test that is done to identify allergens. A suspected substance is applied to the skin. After 24-48 hours, if the area is red and swollen, the test is positive for that substance. If no reaction occurs, another substance is applied. This is continued until the patient experiences an allergic reaction where the irritant was applied to the skin.
— A spotted, pink or red skin eruption that may be accompanied by itching and is caused by disease, contact with an allergen, food ingestion, or drug reaction.
— An open sore on the skin, resulting from tissue destruction, that is usually accompanied by redness, pain, or infection.
dermatitis /der·ma·ti·tis/ (der″mah-ti´tis) pl. dermati´tides inflammation of the skin.
actinic dermatitis dermatitis due to exposure to actinic radiation, such as that from the sun, ultraviolet waves, or x- or gamma radiation.
allergic contact dermatitis contact dermatitis due to allergic sensitization.
ammonia dermatitis diaper dermatitis attributed to skin irritation, due to the ammonia decomposition products of urine.
atopic dermatitis a chronic inflammatory, pruritic, eczematous skin disorder in individuals with a hereditary predisposition to cutaneous pruritus; often accompanied by allergic rhinitis, hay fever, and asthma.
berlock dermatitis , berloque dermatitis dermatitis of the neck, face, or chest, with patches or streaks, caused by exposure to perfume or other toilet articles containing bergamot oil and then to sunlight.
contact dermatitis acute or chronic dermatitis caused by substances contacting the skin; it may involve allergic or nonallergic mechanisms.
exfoliative dermatitis virtually universal erythema, desquamation, scaling, and itching of the skin, with loss of hair.
dermatitis herpetifor´mis pruritic chronic dermatitis with successive groups of symmetrical, erythematous, papular, vesicular, eczematous, or bullous lesions, usually associated with asymptomatic gluten-sensitive enteropathy.
infectious eczematous dermatitis a pustular eczematoid eruption arising from a primary lesion that is the source of an infectious exudate.
insect dermatitis a transient skin eruption caused by the toxin-containing irritant hairs of insects such as certain moths and their caterpillars.
irritant dermatitis a nonallergic type of contact dermatitis due to exposure to a substance that damages the skin.
livedoid dermatitis local pain, swelling, livedoid changes, and increased temperature; due to temporary or prolonged local ischemia from vasculitis or from accidental arterial obliteration during intragluteal administration of medications.
, meadow-grass dermatitis phytophotodermatitis
with eruption of vesicles and bullae in streaks or other configurations, caused by exposure to sunlight after contact with meadow grass.
photoallergic contact dermatitis , photocontact dermatitis allergic contact dermatitis caused by the action of sunlight on skin sensitized by contact with substances such as halogenated salicylanilides, sandalwood oil, or hexachlorophene.
phototoxic dermatitis erythema followed by hyperpigmentation of sun-exposed areas of the skin, due to exposure to agents containing photosensitizing substances, such as coal tar and psoralen-containing perfumes, drugs, or plants, and then to sunlight.
poison ivy dermatitis
, poison oak dermatitis
, poison sumac dermatitis
allergic contact dermatitis due to exposure to plants of the genus Rhus,
which contain urushiol, a skin-sensitizing agent.
rat mite dermatitis that due to a bite of the rat-mite, Ornithonyssus bacoti.
seborrheic dermatitis , dermatitis seborrhe´ica chronic pruritic dermatitis with erythema, scaling, and yellow crust on areas such as the scalp, with exfoliation of excessive dandruff.
stasis dermatitis chronic eczematous dermatitis due to venous insufficiency, initially on the inner aspect of the lower leg above the internal malleolus, sometimes spreading over the lower leg, marked by edema, pigmentation, and often ulceration.
see under itch.
dermatitis (der?ma-tit'is ) (-tit'i-dez?) plural.dermatitidesplural.dermatitises [ dermato- + -itis]
An inflammatory rash marked by itching and redness. See: eczema
Dermatitis has many causes, , including contact with skin irritants (such as the oil that causes poison ivy or oak); venous stasis, with edema and vesicle formation near the ankles; habitual scratching, as is found in neurodermatitis; dry skin, as in winter itch; and ultraviolet light, as in photosensitivity reactions.
When a source of dermatitis is identifiable (such as in contact dermatitis due to a detergent or topical cosmetic), the best treatment is to avoid the irritating substance and to cleanse the affected area immediately with mild soap and water. Once skin inflammation is established, topical corticosteroid ointments or systemic steroids (during extreme exacerbations), topical immunomodulator agents (in patients above age 2), weak tar preparations and ultraviolet B light therapy (to increase the thickness of the stratum corneum) and antihistamines may be used, with antibiotics reserved for secondary infections. Dermatologists may prescribe occlusive dressings intermittently to help clear lichenified skin.
The patient should avoid known skin irritants. Tepid baths, cool compresses, and astringents sometimes help relieve inflammation and itch. Moisturizing creams or lotions following bathing help to retain skin moisture, but perfumed products should be avoided. Drug therapy is administered and evaluated for desired effects and adverse reactions. The patient is taught to apply topical medications and is educated about their most common side effects. Scratching is discouraged and the fingernails kept short to limit excoriation. The patient should be made aware that drowsiness may occur with antihistamine use and that driving or operating mechanical equipment should be avoided until the extent of this effect is known. Health care professionals should be careful not to show any negative feelings when touching lesions during assessment or treatment but should follow standard precautions. Skin changes alter body image, and the patient will need assistance in accepting and coping with what he or she may view as disfigurement. Children and adolescents may require and benefit from counseling to help them deal with emotional components of their condition.
A chronic red or eczematous rash, usually on the face or exposed skin surfaces, that typically results from exposure and sensitization to ultraviolet rays. Adults over age 50 may be affected. See: illustration Synonym: photosensitivity dermatitis
allergic contact dermatitisContact dermatitis.
Chronic dermatitis of unknown cause found in patients with a history of allergy. The disease usually begins after the first 2 months of life, and those affected may experience exacerbations and remissions throughout childhood and adulthood. In many cases, there is a family history of allergy or atopy: if both parents have atopic dermatitis, the chances are nearly 80% that their children will have it, as well. Atopic dermatitis is typically found in flexural creases of the body, e.g., the antecubital and popliteal fossae. The skin lesions consist of reddened, cracked, and thickened skin that can become exudative and crusty from scratching. Scarring or secondary infection may occur. Most patients have an elevated level of immunoglobulin E in their serum. See: illustration
The patient should avoid soaps and ointments. Bathing is kept to a minimum, but bath oils may help to prevent drying of the skin. Clothing should be soft textured and should not contain wool. Fingernails should be kept short to decrease damage from scratching. Antihistamines may help reduce itching at night. Heavy exercise should be avoided because it induces perspiration. A nonlipid softening lotion followed by a corticosteroid in a propylene glycol base may effectively treat acute exacerbations; when large areas of the body are involved, oral steroids may be needed. Because of the adverse effects associated with corticosteroids, topical immunosuppressants such as tacrolimus that decrease T cell activity have been developed. Antistaphylococcal antibiotics may be needed to control secondary infection, introduced when scratching causes microfissures in the skin.
berloque dermatitis, berlock dermatitis
A type of phytophotodermatitis with postinflammatory hyperpigmentation at the site of application of perfumes or colognes containing oil of bergamot. Synonym: bergamot phototoxicity.
Inflammation due to heat, as in sunburn, or cold.
cercarial dermatitisSwimmer's itch.
CONTACT DERMATITIS: Allergic reaction to topical anesthetic
Dermatitis due to contact with allergens or an irritating substance. Allergic contact dermatitis is caused by a T-cell–mediated hypersensitivity reaction to natural or synthetic environmental allergens. These combine with skin proteins, altering the normal autoantigens so that new, foreign antigens are created. Nonallergic contact dermatitis, also known as irritative contact dermatitis, is usually caused by exposure to a detergent, soap, or other skin irritant. Synonym: allergic contact dermatitis dermatitis venenata
(1) See: illustration
Skin changes, which appear 4 to 48 hr after exposure, depending on the degree of sensitivity to the allergen, consist of erythema, local edema, and blisters. The blisters may weep in severe cases. Most patients complain of intense itching. Signs and symptoms of the disease usually last 10 to 14 days. Re-exposure to the cause will trigger a relapse.
Tepid baths, cool compresses, topical astringents (such as solutions of aluminum acetate), antihistamines, and corticosteroids all provide some relief.
contagious pustular dermatitis
A cutaneous disease of sheep and goats transmitted to humans by direct contact. The lesion on humans is usually solitary and on the hands, arms, or face. This maculopapular area may progress to a pustule up to 3 cm in diameter and may last 3 to 6 weeks. The etiological agent is Parapoxvirus, , a genus of poxvirus. Synonym: orf
diaper dermatitisDiaper rash.
Generalized dermatitis, often followed by scaling. It may be caused by leukemias or lymphomas that infiltrate the skin; extensive psoriasis; drug reactions (such as vancomycin); allergies, seborrhea, or atopy. The condition is often associated with systemic findings, including lymphadenopathy, hepatic and splenic enlargement, fever, anemia, eosinophilia, and decreases in serum albumin.
When the skin involvement is extensive, the patient may become depressed because of the cosmetic changes.
Therapy is directed at treating the underlying cause.
A self-inflicted irritation or injury to the skin.
A chronic dermatitis characterized by erythematous, papular, vesicular, bullous, or pustular lesions with a tendency to grouping and with intense itching and burning.
It is associated with allergy to gluten and is often found in patients with celiac disease (gluten-sensitive enteropathy).
The lesions develop suddenly and spread peripherally. The disease is variable and erratic, and an attack may be prolonged for weeks or months. Secondary infection may follow trauma to the inflamed areas.
Oral dapsone provides substantial relief of symptoms in a few days. Sulfapyridine also may be used.
dermatitis hiemalisWinter itch.
dermatitis infectiosa eczematoides
A pustular eruption during or following a pyogenic disease.
livedo-like dermatitisNicolau syndrome.
A blistering rash that appears on the exposed skin of hikers, florists, gardeners, and those who work outdoors in bright sunlight. It is a phototoxic reaction caused by exposure to light-sensitizing chemicals in some plants (such as parsley, rue, bergamot, and fig).
dermatitis medicamentosaDrug rash.
A form of dermatitis with pustular lesions.
dermatitis papillaris capillitii
Formation on the scalp and neck of papules interspersed with pustules. The rash ultimately produces scarlike elevations resembling keloids.
photoallergic contact dermatitisPhotoallergy.
photosensitivity dermatitisActinic dermatitis.
poison ivy dermatitis
Dermatitis resulting from irritation or sensitization of the skin by urushiol, the toxic resin of plants of the genus Toxicodendron
). There is no absolute immunity; susceptibility varies greatly, even in the same individual.
Those sensitive to poison ivy may also react to contact with other plants, such as the mango rind and cashew oil. These plants contain chemicals that cross-react with the sap present in poison ivy, poison oak, and poison sumac.
Some time elapses between skin contact with the poison and first appearance of symptoms, varying from a few hours to several days and depending on the sensitivity of the patient and the condition of the skin. Moderate itching or a burning sensation is soon followed by small blisters; later manifestations vary. Blisters usually rupture and are followed by oozing of serum and subsequent crusting.
Some barrier creams have been used to prevent poison ivy dermatitis. They are sprayed on the skin prior to anticipated contact with the plant.
In mild dermatitis, antihistamines and a lotion to relieve itching are usually sufficient. In severe dermatitis, cool, wet dressings or compresses, potassium permanganate baths, and topical corticosteroids are often effective. In some instances intramuscular or oral corticosteroid therapy is used. If plant leaves are burned and the smoke inhaled, or if plant leaves are ingested, the patient should be directed to an emergency care center. Demulcents, fluids, morphine, and a high-protein, low-fat diet may be prescribed.
Prevention is important in those with known sensitivity and in those with no previous contact with or reaction to the plant. Instruction of the patient focuses on helping the patient to recognize the plant, to avoid contact with it, and to wear long-sleeved shirts and long pants in wooded areas. If contact occurs, the patient should wash with soap and water immediately to remove the toxic oil. Contaminated clothing and pets also should be promptly and thoroughly washed because contact with such items may cause poison dermatitis in other members of the household.
Dermatitis that is a direct rather than an allergic response.
Dermatitis due to radiation exposure. Synonym: radioepidermitis; radioepithelitis; radiodermatitis
Contact dermatitis caused by the toxic resin in poison ivy or oak. See: poison ivy dermatitis; Toxicodendron
schistosome dermatitisSwimmer's itch.
An acute or subacute dermatitis of unknown cause, beginning on the scalp and/or face and in skin folds (any area where sebaceous glands are active) and characterized by rounded, irregular, or circinate lesions covered with yellow or brown-gray greasy scales. Synonym: pityriasis capitis
; seborrhea corporis
; seborrhea sicca
The scalp may be dry with abundant grayish branny scales, or oozing and crusted (eczema capitis). The rash may spread to the forehead and postauricular regions. The forehead shows scaly and infiltrated lesions with dark red bases and localized loss of hair. The eyebrows and eyelashes may have dry, dirty white scales. Inflamed skin and scales may be present on the nasolabial folds or the vermilion border of the lips. On the sternal region, the lesions are greasy to the touch. Eruptions may also appear in interscapular, axillary, and genitocrural regions. Cold winter weather may worsen the condition.
When the condition is limited to the scalp, frequent shampooing and use of mild keratolytic agents are indicated. Selenium-containing shampoos are helpful. Generalized seborrheic dermatitis requires careful attention, including scrupulous skin hygiene, frequent washing and shampooing with selenium sulfide suspension to remove scales, keeping the skin as dry as possible, and using dusting powders. Fluorinated corticosteroids may be applied topically to hairless areas, and systemic cortisone preparations may be required. The differential diagnosis includes psoriasis; it should be ruled out and neurologic conditions recognized as possible predisposing factors.
The health care provider explains to the patient that the condition has remissions and exacerbations and that hormone imbalances, nutritional status, infection, and emotional stress influence its course. The patient is taught to apply prescribed corticosteroids to the body and face. Fluorinated corticosteroids should be used with caution near the eyelids, on the face, and in the groin. To avoid developing a secondary Candida yeast infection in body creases or folds, the patient is advised to cleanse these areas carefully, to dry gently but thoroughly, and to ensure that the skin is well aerated. He or she is taught to treat seborrheic scalp conditions (dandruff) with proper and frequent shampooing, alternating two or three different types of shampoo to prevent the development of resistance to a particular product. External irritants and excessive heat and perspiration should be avoided. Rubbing and scratching the skin are discouraged because they prolong exacerbations and increase the risk for secondary infection and excoriation, esp. since scaly, pruritic lesions present in skin areas with high bacteria counts. Oral antibiotics (such as tetracycline) may be prescribed (as for acne vulgaris) in small doses over a prolonged period to reduce bacterial colonization. The patient is advised to take tetracycline at least 1 hr before or 2 hr after meals, since the drug is poorly absorbed with food. The patient also is taught about the adverse effects of the drug (photosensitivity, birth defects, nausea, vomiting, and candidal vaginitis) and their management. Adherence to the treatment regimen is stressed to achieve optimal results. Psychological support or counseling is provided as necessary to deal with related body image concerns.
Eczema of the legs with edema, pigmentation, and sometimes chronic inflammation. It is usually due to impaired return of blood from the legs. Compression stockings help the rash to resolve gradually. See: illustrationillustration
1. Contact dermatitis.
2. Any inflammation caused by local action of various animal, vegetable, or mineral substances contacting the surface of the skin.
A chronic fungal infection of the skin characterized by the formation of wartlike nodules. These may enlarge and form papillomatous structures that sometimes ulcerate.
This condition may be due to one of several fungi, including Hormodendrum pedrosoi or Phialophora verrucosa.
inflammation of the skin. Dermatitis can result from various animal, vegetable and chemical substances, from heat or cold, from mechanical irritation, from certain forms of malnutrition, or from infectious disease.
rare disease in cattle; large ulcers discharging yellow pus or nodules, on lymphatics with local lymph node enlargement.
acute moist dermatitis
a superficial bacterial infection of the skin, usually caused by self-trauma, i.e. scratching, rubbing, biting. In dogs, ectoparasites, otitis, anal sacculitis and pruritic skin diseases are common precipitating causes. Affected skin is moist, weeping, and has a covering of matted haircoat and dried exudate. Staphylococcus spp. are usually present. Called also pyotraumatic dermatitis, 'hot spots'.
allergic contact dermatitis
see allergic contact dermatitis.
allergic inhalant dermatitis contagious pustular dermatitis of sheep coronet dermatitis
part of several infectious mucosal diseases of cattle; also in equine pemphigus.
equine contagious pustular dermatitis equine exfoliative eosinophilic dermatitis
characterized by infiltration of eosinophils and granulomatous inflammation with ulcerative stomatitis and wasting; suspected of being a hypersensitivity to Strongylus equinus larvae.
equine staphylococcal dermatitis
exudative dermatitis of pigs feline miliary dermatitis
a papular, crusting skin disease located predominantly on the back, with varying degrees of pruritus. Ectoparasites, food and drug allergy, and infection by fungi or bacteria are among the many possible causes. Called also scabby cat disease.
feline psychogenic dermatitis
see idopathic hyperesthesia syndrome.
feline solar dermatitis
see solar dermatitis (below).
dermatitis sufficiently severe to affect deep layers of the dermis results in scarring of the skin due to excessive fibrous tissue formation.
moisture, friction and secondary infection in body folds such as facial fold in brachycephalic dog breeds, tail fold in dog breeds with extremely short, often screw, tails, lip fold in spaniel breeds, perivulvar fold in obese bitches, and all over the body in the Shar pei.
Nasal fold dermatitis in a Bulldog. By permissionfrom Kummel BA, Color Atlas of Small Animal Dermatology,Mosby, 1989
grain itch mite dermatitis granular dermatitis
chronic dermatitis in humans marked by successive crops of grouped, symmetrical, erythematous, papular, vesicular, eczematous or bullous lesions, accompanied by itching and burning; a granular deposition of IgA immunoglobulin around the lesion almost always occurs. Occurs rarely in dogs.
idiopathic caprine dermatitis
alopecic, exudative dermatitis of pygmy goats.
infectious dermatitis of piglets interface dermatitis
a histopathological pattern of inflammatory skin disease with the dermoepidermal junction obscured by hydropic degeneration and/or lichenoid cellular infiltrate.
see fold dermatitis (above).
intraepidermal pustular dermatitis lipfold dermatitis
see fold dermatitis (above).
a pruritic, seborrheic skin disease of dogs, particularly some breeds including West Highland white terriers, Shetland sheepdogs, Poodles, and Cocker spaniels, and rarely cats, caused by colonization of the skin by the yeast, Malassezia pachydermatis. There is usually an underlying cause such as atopy or bacterial pyoderma.
mammary pustular dermatitis
see mammary pustular dermatitis.
an eruption or solitary skin lesion caused by a drug taken internally.
see feline miliary dermatitis (above).
moist dermatitis of rabbits
the rabbit's pendulous dewlap keeps getting wet and develops a moist dermatitis as a result. Called also slobbers, wet dewlap.
nasal solar dermatitis
see solar dermatitis (below), collie nose.
ovine interdigital dermatitis ovine staphylococcal dermatitis
ulcerative dermatitis of the face of adult sheep and young lambs caused by a dermatopathic strain of Staphylococcus aureus. Called also ovine staphylococcal pyoderma.
caused by larvae of the free-living nematode Pelodera strongyloides and characterized by alopecia, itching, thick, scurfy skin and 0.5 inch diameter pustules which contain the larvae.
allergic contact dermatitis caused by the action of sunlight on skin sensitized by contact with a substance capable of causing this reaction.
see photosensitive dermatitis.
plastic dish dermatitis
a contact dermatitis caused by plastic feeding dishes to which a dog is allergic.
porcine juvenile pustular psoriasiform dermatitis primary-irritant dermatitis
contact dermatitis (see above) induced by a substance acting as an irritant rather than as a sensitizer or allergen.
psoriaform, psoriasiform dermatitis of swine pyotraumatic dermatitis
see acute moist dermatitis (above).
a pruritic, papulocrustous dermatitis in dairy cows which is accompanied by fever and hemorrage from the nose and anus. It is believed to be caused by a toxin.
see pelodera dermatitis (above).
seasonal allergic dermatitis
, equine allergic dermatitis
seborrheic dermatitis, dermatitis seborrheica
a chronic, usually pruritic, dermatitis with erythema, dry, moist or greasy scaling, and yellow crusted patches on various areas, with exfoliation of an excessive amount of dry scales (dandruff) or encrustations of sebum on the skin. See also exudative
epidermitis (pigs), greasy heel
a chronic, inflammatory reaction on white or lightly pigmented and exposed skin caused by sunlight. Most commonly seen on the ear tips, nose and eyelids of white cats and the nose of collie dogs or related breeds. Squamous cell carcinomas sometimes develop in affected skin. Called also nasal solar dermatitis, actinic dermatitis. See also collie nose.
perivascular inflammation with spongiosis.
superficial pustular dermatitis
immature dogs may develop pustules on the inguinal or axillary skin, often in association with poor nutrition, systemic infection, or parasitism. In kittens, these may occur on the neck, caused by 'mouthing' by the queen.
unilateral papular dermatitis
a disease of horses characterized by the appearance of many nodules or papules on one side of the neck and body. The lesions are eosinophilic folliculitis and perifolliculitis. The etiology and the unilateral distribution of the lesions are unexplained.
ventral midline dermatitis
small ulcers with hemorrhagic crusts and hair loss, located on the abdomen, particularly around the umbilicus, of horses; caused by biting flies and gnats.
see avian vesicular dermatitis.
viral contagious dermatitis viral papular dermatitis
see equine papular dermatitis.