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chemotaxis
(redirected from Chemotactic factors)

   Also found in: Dictionary/thesaurus, Encyclopedia, Wikipedia 0.01 sec.
chemotaxis /che·mo·tax·is/ (-tak´sis) taxis in response to the influence of chemical stimulation.chemotac´tic
che·mo·tax·is (km-tkss)
n.
The characteristic movement or orientation of an organism or cell along a chemical concentration gradient either toward or away from the chemical stimulus. Also called chemotropism.

chemotaxis
[-tak′sis]
Etymology: Gk, chemeia + taxis, arrangement
movement toward or away from a chemical stimulus. Chemotaxis is a cellular function, particularly of neutrophils and monocytes, whose phagocytic activity is influenced by chemical factors released by invading microorganisms.

chemotaxis [ke″mo-tak´sis]
list; movement (taxis) in response to the influence of chemical stimulation. adj., adj chemotac´tic.
leukocyte chemotaxis the response of leukocytes to products formed in immunologic reactions, wherein leukocytes are attracted to and accumulate at the site of the reaction; a part of the inflammatory response. See also inflammation.

chemotaxis (kē´mōtak´sis),
n a response involving movement that is positive (toward) or negative (away from) to a chemical stimulus.
chemotaxis, leukocyte,
n the phagocytic activity of neutrophils and monocytes in response to chemical factors released by invading microorganisms.

chemotaxis
taxis or directional movement in response to the influence of chemical stimulation.

leukocyte chemotaxis
the response of leukocytes to products formed in immunological reactions, wherein leukocytes are attracted to and accumulate at the site of the reaction; a part of the inflammatory response. See also inflammation.


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Some of the mediators are histamine, heparin, leukotrienes, serotonin, chemotactic factors, tryptase, chymase and prostaglandins.
They can cause vessel dilation, release chemotactic factors, result in extravasation of white blood cells, and dermatologically produce pruritis, erythema, scaling and hyperkeratosis of sebaceous follicles.
6) In rabbits, Hashimoto et al showed that steroids suppress the formation of granulation tissue (1) by reducing vascular permeability, (2) by decreasing extravasation and the migration of inflammatory cells to the site of infection, (3) by inhibiting the production of a variety of chemotactic factors (especially complement factors and lymphocyte-derived factors), and (4) by acting on growth factors such as vascular endothelial growth factor and transforming growth factor beta.
 
 
 
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