Borrelia hermsii

Bor·rel·i·a herm·si·i

a bacterial species found as a cause of relapsing fever in British Columbia, California, Colorado, Idaho, Nevada, Oregon, and Washington; transmitted by a tick, Ornithodoros hermsi.

Borrelia hermsii

The species of gram-negative spirochetes which may cause endemic tick-borne or epidemic louse-borne relapsing fever.
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In North America, this zoonosis is associated with 3 species of spirochetes, but most human cases are caused by Borrelia hermsii, which is found in scattered foci in the western United States and southern British Columbia, Canada (5,6).
Of 39 attendees, 14 (36%) reported fever and at least one of several symptoms associated with the illness (chills, diaphoresis, headache, myalgia, arthralgia, rash, or tick bite) within 7 days after the event; 11 of those had laboratory confirmation of Borrelia hermsii infection (MMWR 52[34]:809-12, 2003).
Reinfection of vervet monkeys (Cercopithecus aethiops) with Borrelia hermsii.
DNA was extracted from the infected liver, and PCR-DNA sequencing of the 16S ribosomal RNA (rRNA) locus identified the bacterium as a relapsing fever spirochete related most closely to Borrelia hermsii (1).
hermsi tick-associated spirochete Spirochaeta hermsi (7), now recognized as Borrelia hermsii (8).
Human disease occurs in many focal areas and is associated with infections of Borrelia hermsii, B.
Borrelia hermsii is the most common cause of tickborne relapsing fever in North America.
Identification and characterization of a linear-plasmid-encoded factor H-binding protein (FhbA) of the relapsing fever spirochete Borrelia hermsii.
Partial sequencing of the 16S-23S rDNA intergenic spacer showed two to four genotypes each for Borrelia hermsii and B.
Borrelia hermsii was isolated from the blood of two patients, and Ornithodoros hermsi ticks were collected from the cabin, the first demonstration of this bacterium and tick in Montana.
Population structure of the relapsing fever spirochete Borrelia hermsii as indicated by polymorphism of two multigene families that encode immunogenic outer surface lipoproteins.