aspartate transaminase

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aspartate transaminase

 (AST) (ASAT) [ah-spahr´tāt trans-am´ĭ-nās]
an enzyme that catalyzes the reversible transfer of an amino group from aspartate to α-ketoglutarate to form glutamate and oxaloacetate, requiring the coenzyme pyridoxal phosphate; it is normally present in serum and in various body tissues, especially in the heart and liver. (See accompanying table.) It is released into the serum as the result of tissue injury, especially injury to the heart or liver, hence the concentration in the serum may be increased in myocardial infarction or acute damage to hepatic cells. Serum levels are also increased in some muscle diseases, such as progressive muscular dystrophy. Called also glutamic-oxaloacetic transaminase.

as·par·tate a·mi·no·trans·fer·ase (AST),

an enzyme catalyzing the reversible transfer of an amine group from l-glutamate to oxaloacetate, forming α-ketoglutarate and l-aspartate; an aid in diagnosing viral hepatitis and myocardial infarction.

aspartate transaminase

/as·par·tate trans·am·i·nase/ (AST) (ASAT) (trans-am´ĭ-nās) an enzyme normally present in body tissues, especially in the heart and liver; it is released into the serum as the result of tissue injury, hence the concentration in the serum (SGOT) may be increased in disorders such as myocardial infarction or acute damage to hepatic cells.

aspartate transaminase

aspartate

Asp; any salt of aspartic acid; aspartic acid in dissociated form.

aspartate carbamoyl transferase
enzyme catalyzing the condensation of carbamoyl phosphate and l-aspartate to N-carbamoyl-l-aspartate. A regulatory, allosteric enzyme in the synthesis of pyrimidine nucleotides.
aspartate transaminase

Patient discussion about aspartate transaminase

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References in periodicals archive ?
Table 1: Blood chemistry and Hematological findings Variable(Unit)(normal range) Value First admission Aspartate amino transferase (U/L)(2-31) 20 Alonine minotransferase (U/L)(2-31) 9 Alkaline phoshatase(U/L)(46-306) 245 Billirubin Total (mg%) (0.
The serum separated was used for assay of Alanine amino transferase (ALT), Aspartate amino transferase (AST), alkaline phosphatase (ALP), albumin, triglycerides and serum bilirubin according to the instructions provided by the company.
Serum alanine amino transferase (ALT) and aspartate amino transferase (AST) activities were determined using Randox assay kits based on the method of Reitman and Frankel (1957).
The following blood parameters were measured: glucose, cholesterol, triglycerides, high-density lipoprotein, cholesterol, uric acid, creatine kinase, aspartate amino transferase, alanine-aminotransferase, alkaline phosphatase, amylase, and pancreatic amylase.
Fenofibrate significantly reduced both liver enzymes, alanine amino transferase (ALT) and aspartate amino transferase (AST), unlike GFT505.
At the end of this period, when compared to controls, it was found that Natrum sulphuricum 200C corrected the levels of acid phosphatase, alkaline phosphatase, aspartate amino transferase, and alanine amino transferase as well as produced an increase in glutathione levels, reduced lipid peroxidation, and reduced the level of damage caused to genes by carcinogens.
The alamine amino transferase and aspartate amino transferase were estimated by the method of Reitman and Fraenkel (1957) as given by Bergmeyer (1965).
The activities of Aspartate Amino Transferase and Alanine Amino Transferase were enhanced over controls in all the tissues, at all the exposure spans.
His liver function tests showed 60- and 70-fold increases in aspartate amino transferase and alanine aminotransferase levels, respectively.
The woman's values were aspartate amino transferase (ASAT) 48 IU/L (normal <26); alanine amino transferase (ALAT) 29 IU/L (normal <27); gamma glutamyl transferase (g-GT) 32 IU/L (normal <200); and lactate dehydrogenase (LDH) 517 IU/L (normal <250).
The most common adverse events irrespective of causality were nausea (38 percent), diarrhea (33 percent) and pyrexia (29 percent); the most common Grade 3 or higher adverse events were febrile neutropenia (25 percent), increased aspartate amino transferase (17 percent) and pneumonia (13 percent)
Alkalin phosphatase levels (Figure 2) and fasting glucose (Figure 5) in all groups and aspartate amino transferase (Figure 1) in diabetic treated groups increased significantly compared to the control group ([?