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Persistent organic pollutants modify gut microbiota- host metabolic homeostasis in mice through aryl hydrocarbon receptor activation.
A role for the aryl hydrocarbon receptor in mammary gland tumorigenesis.
Identification of the aryl hydrocarbon receptor target gene TiPARP as a mediator of suppression of hepatic gluconeogenesis by 2,3,7,8-tetrachlorodibenzo-p-dioxin and of nicotinamide as a corrective agent for this effect.
Induction of CYPl A2 is regulated by aryl hydrocarbon receptor (AhR) which mediates CYPl A2 gene transcription through dioxin response element.
5) The compound has been shown in human and animal studies to alter transcription of genes by binding to the aryl hydrocarbon receptor (AhR).
Polycyclic aromatic hydrocarbons are toxic environmental pollutants, which indirectly act as anti-estrogens by activating the aryl hydrocarbon receptor (AhR).
Within every cell of the body resides a protein known to scientists as the aryl hydrocarbon receptor.
2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) is a ubiquitous environmental pollutant and the most potent aryl hydrocarbon receptor (AHR) ligand.
Most biological effects of TCDD are mediated by the aryl hydrocarbon receptor (AHR), a ligand-activated transcription factor and a member of the basic-region-helix-loop-helix PER/ARNT/SIM (Bhlh-PAS) superfamily of transcription factors.
Most effects of TCDD are mediated through the aryl hydrocarbon receptor (AhR) (Hankinson 1995; Okey et al.
Dioxin is thought to exert its biological and toxicological effects primarily by binding to the aryl hydrocarbon receptor (AhR, dioxin receptor) followed by nuclear translocation and binding to dioxin-responsive elements (DREs) in gene promoters (reviewed by Beischlag et al.
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