Anaplasma phagocytophilum

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A·na·plas·ma pha·go·cy·to·phil·um

(an'ă-plaz'mă fā'gō-sī-tŏ-f'il-um)
A bacterial species that causes human granulocytic ehrlichiosis; also causes tick-borne fever in cattle; spread by ticks (Ixodes), it occurs in the United States in the Middle Atlantic states, southern New England, and the lower Midwest.

Anaplasma phagocytophilum

(an″ă-plaz′mă fag″ŏ-sī-tof′ĭ-lŭm)
A small gram-negative coccus that is an obligate intracellular parasite. It can be transmitted to humans by tick bite and is the cause of the disease formerly known as human granulocytic ehrlichiosis (now known as anaplasmosis). It was formerly called Ehrlichia phagocytophila.


a genus of organisms in the family Anaplasmataceae, order Rickettsiales. Members parasitize erythrocytes, thrombocytes and leukocytes and are transmitted by ticks.

Anaplasma bovis
causes benign bovine rickettsiosis in Asia and Africa. Previously Ehrlichia bovis.
Anaplasma caudatum
often found in mixed infections with A. marginale in cattle.
Anaplasma centrale
causes a mild form of anaplasmosis in cattle and has been used as a vaccine against A. marginale.
Anaplasma marginale
a significant pathogen, the cause of anaplasmosis in ruminants. The infection is transmitted mechanically by many biting insects. Ticks, including Boophilus spp. and Dermacentor spp., are biological vectors.
Anaplasma ovis
the cause of anaplasmosis of sheep.
Anaplasma phagocytophilum
causes tickborne fever or 'pasture disease' in cattle, goats, sheep and wild ruminants; granulocytic ehrlichiosis in cattle, cats and llamas, both of which are characterized by leukopenia and thrombocytopenia, equine ehrlichiosis, and human granulocytic ehrlichiosis. Transmitted by Ixodes ticks. Previously Ehrlichia equi (Rickettsia phagocytophila and Rickettsia equi), E. phagocytophila and the human granulocytic ehrlichiosis (HGE) agent.
Anaplasma platys
the cause of canine infectious cyclic thrombocytopenia. The only rickettsia known to infect platelets. Thought to be transmitted by Rhipicephalus sanguineus. Previously called Ehrlichia platys.
References in periodicals archive ?
Increasing incidence of Ehrlichia chaffeensis and Anaplasma phagocytophilum in
For example, Ixodes ticks can simultaneously or sequentially infect their hosts with Borrelia burgdorferi, Anaplasma phagocytophilum, and Babesia microti.
There are many diseases that can be spread by ticks but those most evident in sheep in the United Kingdom are: | Tick-borne Fever (TBF) - caused by the rickettsia called Anaplasma phagocytophilum, which spreads into the blood from the bite of an infected tick.
Canine Anaplasma phagocytophilum IFA Kit, sufficient for 2000 determinations,
It also is the vector of Anaplasma phagocytophilum and Babesia microti, causative agents of human anaplasmosis and babesiosis, respectively (Estrada-Pefia and Jongejan, 1999).
This bacterium, Anaplasma phagocytophilum (Ap), secretes a protein that binds with another protein produced by white blood cells, and that connection creates compartments that siphon host-cell nutrients to feed the bacteria, enabling their growth inside the white blood cells.
In the study, researchers reported that a protein called OmpA on the surface of Anaplasma phagocytophilum is important for invading host cells.
Exposure to Anaplasma phagocytophilum and ticks in gray foxes (Urocyon cinereoargenteus) in northern Humboldt County, California.
Some of these include other Borrelia species, Babesia microti, Babesia duncani, Ehrlichia chaffeensis, Anaplasma phagocytophilum, Bartonella henselae, Bartonella quintana, Mycoplasma fermentans and other Mycoplasma species, Rickettsia rickettsii, Coxiella burnetii, Francisella tularensis, viruses such as HHV-6, nematodes, and possibly many other organisms.
Role of migratory birds in introduction and range expansion of Ixodes scapularis ticks and of Borrelia burgdorferi and Anaplasma phagocytophilum in Canada.
Researchers in many different countries and climates explore such topics as RNAi and the study of insect immunity, the Picorna-like Kakugo virus identified in aggressive honeybee workers, encephalitic arboviruses as an emerging and re-emerging problem, neurologic manifestations of West Nile Virus infection, the effects of co-infection with Borrelia Burgdorferi and Anaplasma Phagocytophilum in vector ticks and vertebrate hosts, and the cellular secretion of Sf21 cells upon baculovirus infection.