ameloblast

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ameloblast

 [ah-mel´o-blast]
a cell that takes part in forming dental enamel.

am·e·lo·blast

(am'ĕ-lō-blast),
One of the columnar epithelial cells of the inner layer of the enamel organ of a developing tooth, involved with the formation of enamel matrix.
[Early E. amel, enamel, + G. blastos, germ]

ameloblast

/am·e·lo·blast/ (am´ĕ-lo-blast″) a cell that takes part in forming dental enamel.

ameloblast

[am′ilōblast′]
Etymology: OFr, amel + Gk, blastos, germ
an epithelial cell from which tooth enamel is formed. Also called enamel cell. -ameloblastic, adj.

ameloblast

A columnar epithelial cell that secretes tooth enamel, which has tapering apical surfaces and Tomes processes and which is embedded in the enamel matrix of the tooth.

am·e·lo·blast

(am'ěl'ō-blast)
One of the columnar epithelial cells of the inner layer of the enamel organ of a developing tooth, concerned with the formation of enamel.
[Early E. amel, enamel, + G. blastos, germ]

am·e·lo·blast

(am'ěl'ō-blast)
One of the columnar epithelial cells of the inner layer of the enamel organ of a developing tooth, involved with the formation of enamel matrix.
Synonym(s): enamel cell, enameloblast.
[Early E. amel, enamel, + G. blastos, germ]

ameloblast (am´əlōblast´),

n an epithelial cell associated with the enamel organ that, during tooth development, secretes enamel matrix.
ameloblast atrophy,
n a wasting of or decrease in the epithelial cells, which form tooth enamel; may occur as the result of a deficiency in vitamin A. See also atrophy, periodontal.

ameloblast

a cell which takes part in forming dental enamel.
References in periodicals archive ?
Ameloblasts of the control molars and those with low exposure to amoxicillin exhibited normal patterns of amelogenesis, whereas ameloblasts of molars exposed to amoxicillin showed altered patterns of amelogenesis, which might interfere with mineralisation.
Pits are the result of abrupt and permanent interruption of matrix secretion by compact clusters of ameloblasts.
The mature ameloblasts regulate the final mineralisation of enamel.
Therefore, the effects of any insult to the ameloblasts are detectable as defects in the mature enamel.
It has been suggested that the causative factor of MIH or opacities in molars and incisors, could have been oxygen lack in active ameloblasts [van Amerongen and Kreulen, 1995; Seow, 1996; Aine et al.
Oxygen tension markers showed little variation in mature ameloblasts of the test rat incisors compared with those of the control animals while hypoxic disturbances were observed in the cells of the pulp and surrounding periodontium.
Effects on developing ameloblasts may be important.
Pre-term children, when their ameloblasts are in a secretory stage, usually have hypoplasia and not hypomineralisation which is part of the maturation stage.
An ultrastructural study of the effect of streptozotocin on the secretory ameloblasts of the rat incisor.
Conclusions: Diabetes may have a profound detrimental influence on the function of ameloblasts in laying down enamel.
However, experimentally induced diabetes has been shown to inhibit enamel protein secretion by secretory ameloblasts [Karim, 1983].
Effects of a macrolide antibiotic on enamel formation in rat incisors--primary lesion of ameloblast at the transition stage.